TSPY10

Chr Y

testis specific protein Y-linked 10

The protein is predicted to bind chromatin and histones and may be involved in sperm differentiation and proliferation. No established Mendelian diseases are currently associated with TSPY10 mutations in pediatric patients. This gene shows low constraint against loss-of-function variants, suggesting tolerance to such changes.

ResearchSummary from RefSeq, UniProt
GOFmechanismLOEUF 1.90
Clinical SummaryTSPY10
Population Constraint (gnomAD)
Constrained for loss-of-function variants (OE-LoF 0.00) despite low pLI — interpret in context.

Population Genetics & Constraint

gnomAD v4 — loss-of-function & missense intolerance

Tolerant — LoF & missense variants common in population
LoF Constraint
1.90LOEUF
pLI 0.284
Z-score 0.17
OE 0.00 (0.001.90)
Tolerant

Highly tolerant — LoF variants common in population

Missense Constraint
0.16Z-score
OE missense 0.00 (0.001.88)
0 obs / 0.2 exp
Tolerant

Mild missense constraint

Observed / Expected Ratios
LoF OE0.00 (0.001.90)
00.351.4
Missense OE0.00 (0.001.88)
00.61.4
Synonymous OE0.00
01.21.6
LoF obs/exp: 0 / 0.0Missense obs/exp: 0 / 0.2Syn Z: 0.25
DN
0.6065th %ile
GOF
0.73top 25%
LOF
0.2190th %ile

The highest-scoring mechanism for this gene is gain-of-function.

GOFprediction above median

Note: In-silico variant effect predictors (SIFT, PolyPhen, REVEL, CADD) may underestimate pathogenicity of missense variants in genes with GOF or DN mechanisms. Consider functional evidence and clinical context.

Predictions from Badonyi M, Marsh JA. PLoS ONE. 2024;19(8):e0307312.

ClinVar Variant Classifications

0 submitted variants in ClinVar

Protein Context — Lollipop Plot

TSPY10 · protein map & ClinVar variants

Showing all ClinVar variants across the protein. Search a specific variant to highlight its position.

3D Protein StructureAlphaFold

Clinical Trials

Active and recruiting trials from ClinicalTrials.gov

No active trials found for this gene.

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Clinical Literature
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