TSEN2

Chr 3AR

tRNA splicing endonuclease subunit 2

Also known as: PCH2B, SEN2, SEN2L

The protein is a subunit of tRNA splicing endonuclease that catalyzes the removal of introns during the first step of RNA splicing. Mutations cause pontocerebellar hypoplasia type 2B, an autosomal recessive neurodegenerative disorder. The pathogenic mechanism involves impaired tRNA processing leading to disrupted protein synthesis.

OMIMResearchSummary from RefSeq, OMIM, UniProt
ARLOEUF 1.141 OMIM phenotype
Clinical SummaryTSEN2
Population Constraint (gnomAD)
Low constraint (pLI 0.00) — loss-of-function variants are relatively tolerated in the population.

Population Genetics & Constraint

gnomAD v4 — loss-of-function & missense intolerance

Tolerant — LoF & missense variants common in population
LoF Constraint
1.14LOEUF
pLI 0.000
Z-score 1.00
OE 0.79 (0.551.14)
Tolerant

Highly tolerant — LoF variants common in population

Missense Constraint
-1.00Z-score
OE missense 1.17 (1.071.29)
308 obs / 262.5 exp
Tolerant

Tolerant to missense variation

Observed / Expected Ratios
LoF OE0.79 (0.551.14)
00.351.4
Missense OE1.17 (1.071.29)
00.61.4
Synonymous OE1.04
01.21.6
LoF obs/exp: 20 / 25.4Missense obs/exp: 308 / 262.5Syn Z: -0.34

ClinVar Variant Classifications

0 submitted variants in ClinVar

Protein Context — Lollipop Plot

TSEN2 · protein map & ClinVar variants

Showing all ClinVar variants across the protein. Search a specific variant to highlight its position.

3D Protein StructureAlphaFold

Clinical Trials

Active and recruiting trials from ClinicalTrials.gov

No active trials found for this gene.

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Clinical Literature
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Recent Gene-Specific Literature
Gene in title · MEDLINE · newest first
Europe PMC