TRDMT1

Chr 10

tRNA aspartic acid methyltransferase 1

Also known as: DMNT2, DNMT2, MHSAIIP, PUMET, RNMT1

This protein specifically methylates cytosine-38 in the anticodon loop of aspartic acid transfer RNA (tRNA-Asp), with higher activity on tRNA modified with queuosine. Mutations cause autosomal recessive intellectual developmental disorder with cardiac arrhythmias, which typically presents in early childhood with developmental delays and cardiac conduction abnormalities. The gene shows very high constraint against loss-of-function variants, indicating that complete loss of protein function is poorly tolerated.

Summary from RefSeq, UniProt
Research Assistant →
0
Active trials
14
Pubs (1 yr)
14
P/LP submissions
0%
P/LP missense
1.19
LOEUF
DN
Mechanism· predicted
Clinical SummaryTRDMT1
Population Constraint (gnomAD)
Low constraint (pLI 0.00) — loss-of-function variants are relatively tolerated in the population.
📋
ClinVar Variants
14 unique Pathogenic / Likely Pathogenic· 58 VUS of 101 total submissions

Population Genetics & Constraint

gnomAD v4 — loss-of-function & missense intolerance

Tolerant — LoF & missense variants common in population
LoF Constraint
1.19LOEUF
pLI 0.000
Z-score 0.88
OE 0.79 (0.541.19)
Tolerant

Highly tolerant — LoF variants common in population

Missense Constraint
-0.82Z-score
OE missense 1.17 (1.051.30)
224 obs / 192.0 exp
Tolerant

Tolerant to missense variation

Observed / Expected Ratios
LoF OE0.79 (0.541.19)
00.351.4
Missense OE1.17 (1.051.30)
00.61.4
Synonymous OE1.18
01.21.6
LoF obs/exp: 17 / 21.4Missense obs/exp: 224 / 192.0Syn Z: -1.16
DN
0.6647th %ile
GOF
0.4578th %ile
LOF
0.2485th %ile

The highest-scoring mechanism for this gene is dominant-negative.

DNprediction above median

Note: In-silico variant effect predictors (SIFT, PolyPhen, REVEL, CADD) may underestimate pathogenicity of missense variants in genes with GOF or DN mechanisms. Consider functional evidence and clinical context.

Predictions from Badonyi M, Marsh JA. PLoS ONE. 2024;19(8):e0307312.

ClinVar Variant Classifications

101 submitted variants in ClinVar

Classification Summary

Pathogenic14
VUS58
Likely Benign3
Benign3
14
Pathogenic
58
VUS
3
Likely Benign
3
Benign

Curated Variants Distribution

Classified variants from ClinVar · 5 ACMG categories

ClassificationLoFMissense + InframeNon-codingSynonymousTotal
Pathogenic
0
0
14
0
14
Likely Pathogenic
0
0
0
0
0
VUS
0
52
6
0
58
Likely Benign
0
3
0
0
3
Benign
0
1
1
1
3
Total05621178

LoF = frameshift, stop gained/lost, canonical splice · Counts from ClinVar esearch · Updated hourly

View in ClinVar →

Protein Context — Lollipop Plot

TRDMT1 · protein map & ClinVar variants

Showing all ClinVar variants across the protein. Search a specific variant to highlight its position.

Clinical Trials

Active and recruiting trials from ClinicalTrials.gov

No active trials found for this gene.

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Clinical Literature
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