TMED9

Chr 5

transmembrane p24 trafficking protein 9

Also known as: GMP25, HSGP25L2G, p24a2, p24alpha2, p25

The TMED9 protein functions in vesicular protein trafficking within the early secretory pathway, particularly in COPI vesicle-mediated retrograde transport and retention of protein complexes in cis-Golgi membranes. Mutations in TMED9 cause autosomal recessive intellectual disability with hypotonia and dysmorphic features, typically presenting in early childhood. The gene shows low constraint to loss-of-function mutations, which is consistent with the recessive inheritance pattern observed in affected patients.

Summary from RefSeq, UniProt
Research Assistant →
0
Active trials
6
Pubs (1 yr)
56
P/LP submissions
0%
P/LP missense
0.82
LOEUF
DN
Mechanism· predicted
Clinical SummaryTMED9
Population Constraint (gnomAD)
Low constraint (pLI 0.01) — loss-of-function variants are relatively tolerated in the population.
📋
ClinVar Variants
55 unique Pathogenic / Likely Pathogenic· 33 VUS of 100 total submissions

Population Genetics & Constraint

gnomAD v4 — loss-of-function & missense intolerance

Tolerant — LoF & missense variants common in population
LoF Constraint
0.82LOEUF
pLI 0.006
Z-score 2.05
OE 0.42 (0.230.82)
Tolerant

Typical tolerance to LoF variation

Missense Constraint
0.93Z-score
OE missense 0.77 (0.660.91)
104 obs / 134.3 exp
Tolerant

Mild missense constraint

Observed / Expected Ratios
LoF OE0.42 (0.230.82)
00.351.4
Missense OE0.77 (0.660.91)
00.61.4
Synonymous OE1.05
01.21.6
LoF obs/exp: 6 / 14.4Missense obs/exp: 104 / 134.3Syn Z: -0.26
DN
0.7327th %ile
GOF
0.5563th %ile
LOF
0.3163th %ile

The highest-scoring mechanism for this gene is dominant-negative.

DNprediction above median

Note: In-silico variant effect predictors (SIFT, PolyPhen, REVEL, CADD) may underestimate pathogenicity of missense variants in genes with GOF or DN mechanisms. Consider functional evidence and clinical context.

Predictions from Badonyi M, Marsh JA. PLoS ONE. 2024;19(8):e0307312.

ClinVar Variant Classifications

100 submitted variants in ClinVar

Classification Summary

Pathogenic50
Likely Pathogenic5
VUS33
Likely Benign2
50
Pathogenic
5
Likely Pathogenic
33
VUS
2
Likely Benign

Curated Variants Distribution

Classified variants from ClinVar · 5 ACMG categories

ClassificationLoFMissense + InframeNon-codingSynonymousTotal
Pathogenic
0
0
50
0
50
Likely Pathogenic
0
0
5
0
5
VUS
0
22
11
0
33
Likely Benign
0
1
1
0
2
Benign
0
0
0
0
0
Total02367090

LoF = frameshift, stop gained/lost, canonical splice · Counts from ClinVar esearch · Updated hourly

View in ClinVar →

Protein Context — Lollipop Plot

TMED9 · protein map & ClinVar variants

Showing all ClinVar variants across the protein. Search a specific variant to highlight its position.

Clinical Trials

Active and recruiting trials from ClinicalTrials.gov

No active trials found for this gene.

Search ClinicalTrials.gov →
Clinical Literature
Open Research Assistant →