TIMM50

Chr 19AR

translocase of inner mitochondrial membrane 50

Also known as: MGCA9, TIM50, TIM50L

This protein is an essential component of the TIM23 complex that translocates proteins across the mitochondrial inner membrane by recognizing mitochondrial targeting signals on incoming proteins. Mutations cause 3-methylglutaconic aciduria type IX with autosomal recessive inheritance. The gene shows moderate constraint against loss-of-function variants (LOEUF 0.646), consistent with mitochondrial dysfunction when both copies are disrupted.

OMIMResearchSummary from RefSeq, OMIM, UniProt
ARLOEUF 0.651 OMIM phenotype
Clinical SummaryTIMM50
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Gene-Disease Validity (ClinGen)
3-methylglutaconic aciduria type 9 · ARDefinitive

Definitive — sufficient evidence for diagnostic panels

Population Constraint (gnomAD)
Low constraint (pLI 0.00) — loss-of-function variants are relatively tolerated in the population.

Population Genetics & Constraint

gnomAD v4 — loss-of-function & missense intolerance

Tolerant — LoF & missense variants common in population
LoF Constraint
0.65LOEUF
pLI 0.002
Z-score 2.88
OE 0.37 (0.220.65)
Tolerant

Typical tolerance to LoF variation

Missense Constraint
0.04Z-score
OE missense 0.99 (0.901.09)
291 obs / 293.0 exp
Tolerant

Mild missense constraint

Observed / Expected Ratios
LoF OE0.37 (0.220.65)
00.351.4
Missense OE0.99 (0.901.09)
00.61.4
Synonymous OE1.13
01.21.6
LoF obs/exp: 9 / 24.3Missense obs/exp: 291 / 293.0Syn Z: -1.15

ClinVar Variant Classifications

0 submitted variants in ClinVar

Protein Context — Lollipop Plot

TIMM50 · protein map & ClinVar variants

Showing all ClinVar variants across the protein. Search a specific variant to highlight its position.

3D Protein StructureAlphaFold

Clinical Trials

Active and recruiting trials from ClinicalTrials.gov

No active trials found for this gene.

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Clinical Literature
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