TIGIT

Chr 3

T cell immunoreceptor with Ig and ITIM domains

Also known as: VSIG9, VSTM3, WUCAM

TIGIT encodes an inhibitory immune receptor that suppresses T-cell and NK cell activation by binding to ligands PVR/CD155 and NECTIN2/CD112 on antigen-presenting cells, thereby modulating immune responses through inhibition of PI3K, MAPK, and NF-kappa-B signaling pathways. The gene shows low constraint to loss-of-function variation (pLI 0.02, LOEUF 1.05), and no Mendelian diseases have been established from mutations in this gene. TIGIT is primarily studied in the context of cancer immunotherapy and autoimmune diseases rather than pediatric neurogenetic disorders.

OMIMResearchSummary from RefSeq, UniProt
MultiplemechanismLOEUF 1.05
Clinical SummaryTIGIT
Population Constraint (gnomAD)
Low constraint (pLI 0.02) — loss-of-function variants are relatively tolerated in the population.
💊
Clinical Trials
6 active or recruiting trials — potential therapeutic options may be available

Population Genetics & Constraint

gnomAD v4 — loss-of-function & missense intolerance

Tolerant — LoF & missense variants common in population
LoF Constraint
1.05LOEUF
pLI 0.018
Z-score 1.48
OE 0.46 (0.231.05)
Tolerant

Highly tolerant — LoF variants common in population

Missense Constraint
-0.04Z-score
OE missense 1.01 (0.881.16)
143 obs / 141.6 exp
Tolerant

Tolerant to missense variation

Observed / Expected Ratios
LoF OE0.46 (0.231.05)
00.351.4
Missense OE1.01 (0.881.16)
00.61.4
Synonymous OE0.90
01.21.6
LoF obs/exp: 4 / 8.7Missense obs/exp: 143 / 141.6Syn Z: 0.61
DN
0.83top 10%
GOF
0.77top 25%
LOF
0.1697th %ile

This gene has evidence for multiple mechanisms of pathogenicity (dominant-negative and gain-of-function). Both the Badonyi & Marsh prediction and the broader genomic evidence point to dominant-negative as the predominant mechanism. Different variants in this gene may act through different mechanisms — interpret in context of the specific variant.

DNprediction above median
GOFprediction above median

Note: In-silico variant effect predictors (SIFT, PolyPhen, REVEL, CADD) may underestimate pathogenicity of missense variants in genes with GOF or DN mechanisms. Consider functional evidence and clinical context.

Predictions from Badonyi M, Marsh JA. PLoS ONE. 2024;19(8):e0307312.

ClinVar Variant Classifications

0 submitted variants in ClinVar

Protein Context — Lollipop Plot

TIGIT · protein map & ClinVar variants

Showing all ClinVar variants across the protein. Search a specific variant to highlight its position.

3D Protein StructureAlphaFold
Clinical Literature
Open Research Assistant →
Full-Text Mentions
NLP-detected gene mentions in article bodies · via PubTator3
PubTator3
Top 5 full-text resultsSearch PubTator3 ↗
Key Publications
Landmark & review papers · by relevance
PubMed
Top 5 results · since 2015Search PubMed ↗