SUV39H2

Chr 10

SUV39H2 histone lysine methyltransferase

Also known as: KMT1B

SUV39H2 encodes a histone methyltransferase that catalyzes trimethylation of histone H3 lysine 9 (H3K9me3), establishing constitutive heterochromatin at pericentric and telomeric regions and mediating epigenetic transcriptional repression. Mutations cause autosomal dominant intellectual disability with developmental delay and behavioral abnormalities. The gene is highly constrained against loss-of-function variants (pLI 0.93, LOEUF 0.36), indicating intolerance to protein disruption.

Summary from RefSeq, UniProt
Research Assistant →
0
Active trials
14
Pubs (1 yr)
18
P/LP submissions
0%
P/LP missense
0.36
LOEUF
Mechanism
Clinical SummarySUV39H2
Population Constraint (gnomAD)
Highly constrained gene — heterozygous loss-of-function variants are very rare in the population (pLI 0.93). One damaged copy is likely sufficient to cause disease.
📋
ClinVar Variants
18 unique Pathogenic / Likely Pathogenic· 26 VUS of 62 total submissions

Population Genetics & Constraint

gnomAD v4 — loss-of-function & missense intolerance

Dual constrained — LoF & missense intolerant
LoF Constraint
0.36LOEUF
pLI 0.929
Z-score 3.72
OE 0.14 (0.060.36)
Highly constrained

More LoF-intolerant than ~75% of genes

Missense Constraint
3.28Z-score
OE missense 0.36 (0.300.44)
76 obs / 209.8 exp
Constrained

Highly missense-constrained (top ~0.1%)

Observed / Expected Ratios
LoF OE0.14 (0.060.36)
00.351.4
Missense OE0.36 (0.300.44)
00.61.4
Synonymous OE0.95
01.21.6
LoF obs/exp: 3 / 21.7Missense obs/exp: 76 / 209.8Syn Z: 0.36

ClinVar Variant Classifications

62 submitted variants in ClinVar

Classification Summary

Pathogenic18
VUS26
Likely Benign5
Benign4
18
Pathogenic
26
VUS
5
Likely Benign
4
Benign

Curated Variants Distribution

Classified variants from ClinVar · 5 ACMG categories

ClassificationLoFMissense + InframeNon-codingSynonymousTotal
Pathogenic
0
0
18
0
18
Likely Pathogenic
0
0
0
0
0
VUS
0
24
2
0
26
Likely Benign
0
1
3
1
5
Benign
0
0
4
0
4
Total02527153

LoF = frameshift, stop gained/lost, canonical splice · Counts from ClinVar esearch · Updated hourly

View in ClinVar →

Protein Context — Lollipop Plot

SUV39H2 · protein map & ClinVar variants

Showing all ClinVar variants across the protein. Search a specific variant to highlight its position.

3D Protein StructureAlphaFold

Clinical Trials

Active and recruiting trials from ClinicalTrials.gov

No active trials found for this gene.

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Clinical Literature
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