Ensembl is currently experiencing issues

The Ensembl REST API is temporarily unavailable. Some gene data (transcript details, protein domains) may be incomplete. Other data sources are unaffected.

You can check Ensembl's status at status.ensembl.org

SPATCCM

Chr 2AR

solute carrier family 1 member 4

Also known as: ASCT1, SATT, SPATCCM

The protein is a sodium-dependent neutral amino acid transporter that facilitates uptake of alanine, serine, cysteine, and threonine. Mutations cause spastic tetraplegia with thin corpus callosum and progressive microcephaly, involving both the central nervous system motor pathways and brain structural development. The condition follows autosomal recessive inheritance.

OMIMResearchSummary from RefSeq, OMIM
AR1 OMIM phenotype
Some data sources returned errors (2)

ensembl: Error: Ensembl fetch failed: 400 for https://rest.ensembl.org/lookup/symbol/homo_sapiens/SPATCCM?content-type=application/json&expand=1

gnomad: Error: Gene not found

Population Genetics & Constraint

Constraint data not available from gnomAD.

ClinVar Variant Classifications

0 submitted variants in ClinVar

Protein Context — Lollipop Plot

SPATCCM · protein map & ClinVar variants

Showing all ClinVar variants across the protein. Search a specific variant to highlight its position.

3D Protein StructureAlphaFold

Clinical Trials

Active and recruiting trials from ClinicalTrials.gov

No active trials found for this gene.

Search ClinicalTrials.gov →
Clinical Literature
Open Research Assistant →
Key Publications
Landmark & review papers · by relevance
PubMed
Dominant-negative variant in SLC1A4 causes an autosomal dominant epilepsy syndrome.
Pujol-Giménez J et al.·Ann Clin Transl Neurol
2023Case report
Top 2 results · since 2015Search PubMed ↗