SPATA31D4

Chr 9

SPATA31 subfamily D member 4

Also known as: FAM75D4

The protein is predicted to be involved in cell differentiation and spermatogenesis and is located in cellular membranes. Clinical disease associations, inheritance patterns, and confirmed pathogenic mechanisms for SPATA31D4 mutations have not been established based on the available information.

Summary from RefSeq, UniProt, Mechanism
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0
Active trials
0
Pubs (1 yr)
0
P/LP submissions
P/LP missense
LOEUF
Multiple*
Mechanism· predicted

Population Genetics & Constraint

Constraint data not available from gnomAD.

DN
0.74top 25%
GOF
0.78top 25%
LOF
0.2971th %ile

This gene has evidence for multiple mechanisms of pathogenicity (gain-of-function and dominant-negative). Both the Badonyi & Marsh prediction and the broader genomic evidence point to gain-of-function as the predominant mechanism. Different variants in this gene may act through different mechanisms — interpret in context of the specific variant.

GOFprediction above median
DNprediction above median

Note: In-silico variant effect predictors (SIFT, PolyPhen, REVEL, CADD) may underestimate pathogenicity of missense variants in genes with GOF or DN mechanisms. Consider functional evidence and clinical context.

Predictions from Badonyi M, Marsh JA. PLoS ONE. 2024;19(8):e0307312.

ClinVar Variant Classifications

0 submitted variants in ClinVar

Protein Context — Lollipop Plot

SPATA31D4 · protein map & ClinVar variants

Showing all ClinVar variants across the protein. Search a specific variant to highlight its position.

Clinical Trials

Active and recruiting trials from ClinicalTrials.gov

No active trials found for this gene.

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Clinical Literature
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Full-Text Mentions
NLP-detected gene mentions in article bodies · via PubTator3
PubTator3
Top 5 full-text resultsSearch PubTator3 ↗
Recent Gene-Specific Literature
Gene in title · MEDLINE · newest first
Europe PMC

No open access results found