SPATA31C2

Chr 9

SPATA31 subfamily C member 2

Also known as: FAM75C2

The SPATA31C2 protein is predicted to be involved in cell differentiation and spermatogenesis and is located in cellular membranes. Currently, no established human diseases have been definitively associated with mutations in this gene. Clinical significance of variants in SPATA31C2 remains to be determined.

ResearchSummary from RefSeq, UniProt
Multiplemechanism

Population Genetics & Constraint

Constraint data not available from gnomAD.

DN
0.76top 25%
GOF
0.79top 10%
LOF
0.2872th %ile

This gene has evidence for multiple mechanisms of pathogenicity (gain-of-function and dominant-negative). Both the Badonyi & Marsh prediction and the broader genomic evidence point to gain-of-function as the predominant mechanism. Different variants in this gene may act through different mechanisms — interpret in context of the specific variant.

GOFprediction above median
DNprediction above median

Note: In-silico variant effect predictors (SIFT, PolyPhen, REVEL, CADD) may underestimate pathogenicity of missense variants in genes with GOF or DN mechanisms. Consider functional evidence and clinical context.

Predictions from Badonyi M, Marsh JA. PLoS ONE. 2024;19(8):e0307312.

ClinVar Variant Classifications

0 submitted variants in ClinVar

Protein Context — Lollipop Plot

SPATA31C2 · protein map & ClinVar variants

Showing all ClinVar variants across the protein. Search a specific variant to highlight its position.

3D Protein StructureAlphaFold

Clinical Trials

Active and recruiting trials from ClinicalTrials.gov

No active trials found for this gene.

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Clinical Literature
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Recent Gene-Specific Literature
Gene in title · MEDLINE · newest first
Europe PMC

No open access results found