SGCG

Chr 13AR

sarcoglycan gamma

Also known as: 35DAG, A4, DAGA4, DMDA, DMDA1, LGMD2C, LGMDR5, MAM

Gamma-sarcoglycan is a sarcolemmal transmembrane glycoprotein that forms part of the dystrophin-glycoprotein complex, providing structural linkage between the muscle fiber cytoskeleton and extracellular matrix. Mutations cause limb-girdle muscular dystrophy type 2C (LGMD2C), an autosomal recessive early-onset muscular dystrophy. The pathogenic mechanism involves loss of protein function leading to sarcolemmal instability and progressive muscle degeneration.

OMIMResearchSummary from RefSeq, OMIM, UniProt, Mechanism
MultiplemechanismARLOEUF 1.051 OMIM phenotype
VCEP Guidelines: Limb Girdle Muscular DystrophyReleased
ClinGen Panel
Clinical SummarySGCG
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Gene-Disease Validity (ClinGen)
autosomal recessive limb-girdle muscular dystrophy · ARDefinitive

Definitive — sufficient evidence for diagnostic panels

Population Constraint (gnomAD)
Low constraint (pLI 0.00) — loss-of-function variants are relatively tolerated in the population.
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Clinical Trials
1 active or recruiting trial — potential therapeutic options may be available

Population Genetics & Constraint

gnomAD v4 — loss-of-function & missense intolerance

Tolerant — LoF & missense variants common in population
LoF Constraint
1.05LOEUF
pLI 0.000
Z-score 1.44
OE 0.60 (0.361.05)
Tolerant

Highly tolerant — LoF variants common in population

Missense Constraint
-0.13Z-score
OE missense 1.03 (0.911.17)
168 obs / 163.2 exp
Tolerant

Tolerant to missense variation

Observed / Expected Ratios
LoF OE0.60 (0.361.05)
00.351.4
Missense OE1.03 (0.911.17)
00.61.4
Synonymous OE1.18
01.21.6
LoF obs/exp: 9 / 15.0Missense obs/exp: 168 / 163.2Syn Z: -1.14
DN
0.81top 10%
GOF
0.7127th %ile
LOF
0.1993th %ile

This gene has evidence for multiple mechanisms of pathogenicity (dominant-negative and gain-of-function). Both the Badonyi & Marsh prediction and the broader genomic evidence point to dominant-negative as the predominant mechanism. Different variants in this gene may act through different mechanisms — interpret in context of the specific variant.

DNprediction above median
GOFprediction above median

Note: In-silico variant effect predictors (SIFT, PolyPhen, REVEL, CADD) may underestimate pathogenicity of missense variants in genes with GOF or DN mechanisms. Consider functional evidence and clinical context.

Predictions from Badonyi M, Marsh JA. PLoS ONE. 2024;19(8):e0307312.

ClinVar Variant Classifications

0 submitted variants in ClinVar

Protein Context — Lollipop Plot

SGCG · protein map & ClinVar variants

Showing all ClinVar variants across the protein. Search a specific variant to highlight its position.

3D Protein StructureAlphaFold
Clinical Literature
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