RPP25L

Chr 9

ribonuclease P/MRP subunit p25 like

Also known as: C9orf23, bA296L22.5

This protein may function as a component of ribonuclease P or MRP, enzymes involved in RNA processing. Mutations in RPP25L cause autosomal recessive microcephaly, short stature, and intellectual disability. The gene shows moderate constraint against loss-of-function variants, suggesting some intolerance to complete protein loss.

ResearchSummary from RefSeq, UniProt
LOEUF 0.81
Clinical SummaryRPP25L
Population Constraint (gnomAD)
Moderately constrained gene (pLI 0.68) — some intolerance to loss-of-function variants.

Population Genetics & Constraint

gnomAD v4 — loss-of-function & missense intolerance

Moderate LoF intolerance
LoF Constraint
0.81LOEUF
pLI 0.676
Z-score 1.78
OE 0.00 (0.000.81)
Moderately constrained

Typical tolerance to LoF variation

Missense Constraint
0.79Z-score
OE missense 0.79 (0.660.94)
87 obs / 110.5 exp
Tolerant

Mild missense constraint

Observed / Expected Ratios
LoF OE0.00 (0.000.81)
00.351.4
Missense OE0.79 (0.660.94)
00.61.4
Synonymous OE0.92
01.21.6
LoF obs/exp: 0 / 3.7Missense obs/exp: 87 / 110.5Syn Z: 0.43

ClinVar Variant Classifications

0 submitted variants in ClinVar

Protein Context — Lollipop Plot

RPP25L · protein map & ClinVar variants

Showing all ClinVar variants across the protein. Search a specific variant to highlight its position.

3D Protein StructureAlphaFold

Clinical Trials

Active and recruiting trials from ClinicalTrials.gov

No active trials found for this gene.

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Clinical Literature
Open Research Assistant →
Full-Text Mentions
NLP-detected gene mentions in article bodies · via PubTator3
PubTator3
Top 5 full-text resultsSearch PubTator3 ↗
Key Publications
Landmark & review papers · by relevance
PubMed
Top 1 results · since 2015Search PubMed ↗
Recent Gene-Specific Literature
Gene in title · MEDLINE · newest first
Europe PMC

No open access results found