RMI2

Chr 16

RecQ mediated genome instability 2

Also known as: BLAP18, C16orf75

RMI2 encodes an essential component of the RMI complex that regulates homologous recombination DNA repair, sister chromatid segregation, and is required for the stability and function of the BLM helicase complex. Mutations cause Bloom syndrome, an autosomal recessive disorder characterized by severe growth deficiency, immunodeficiency, and predisposition to cancer. The gene shows low constraint against loss-of-function variants (pLI 0.019, LOEUF 1.91), consistent with recessive inheritance patterns.

OMIMResearchSummary from RefSeq, UniProt
LOEUF 1.91
Clinical SummaryRMI2
Population Constraint (gnomAD)
Low constraint (pLI 0.02) — loss-of-function variants are relatively tolerated in the population.

Population Genetics & Constraint

gnomAD v4 — loss-of-function & missense intolerance

Tolerant — LoF & missense variants common in population
LoF Constraint
1.91LOEUF
pLI 0.019
Z-score -0.35
OE 1.31 (0.431.91)
Tolerant

Highly tolerant — LoF variants common in population

Missense Constraint
-0.62Z-score
OE missense 1.30 (1.021.66)
45 obs / 34.7 exp
Tolerant

Tolerant to missense variation

Observed / Expected Ratios
LoF OE1.31 (0.431.91)
00.351.4
Missense OE1.30 (1.021.66)
00.61.4
Synonymous OE1.54
01.21.6
LoF obs/exp: 2 / 1.5Missense obs/exp: 45 / 34.7Syn Z: -1.49

ClinVar Variant Classifications

0 submitted variants in ClinVar

Protein Context — Lollipop Plot

RMI2 · protein map & ClinVar variants

Showing all ClinVar variants across the protein. Search a specific variant to highlight its position.

3D Protein StructureAlphaFold

Clinical Trials

Active and recruiting trials from ClinicalTrials.gov

No active trials found for this gene.

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Clinical Literature
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Recent Gene-Specific Literature
Gene in title · MEDLINE · newest first
Europe PMC