RIT2

Chr 18

Ras like without CAAX 2

Also known as: RIBA, RIN, ROC2

RIT2 encodes a small GTPase that binds and exchanges GTP and GDP, and modulates activation of the transcription factor POU4F1. Mutations cause autosomal dominant microcephaly with simplified gyral pattern, abnormal muscle tone, and intellectual disability, typically presenting in infancy. This gene is not highly constrained against loss-of-function variants and has an associated GeneReviews entry for additional clinical guidance.

Summary from RefSeq, UniProt
Research Assistant →
0
Active trials
5
Pubs (1 yr)
45
P/LP submissions
0%
P/LP missense
1.30
LOEUF
Multiple*
Mechanism· predicted
Clinical SummaryRIT2
Population Constraint (gnomAD)
Low constraint (pLI 0.00) — loss-of-function variants are relatively tolerated in the population.
📋
ClinVar Variants
43 unique Pathogenic / Likely Pathogenic· 26 VUS of 79 total submissions
📖
GeneReview available — RIT2
Authoritative clinical overview · Recommended first read
Open GeneReview ↗

Population Genetics & Constraint

gnomAD v4 — loss-of-function & missense intolerance

Tolerant — LoF & missense variants common in population
LoF Constraint
1.30LOEUF
pLI 0.001
Z-score 0.95
OE 0.66 (0.361.30)
Tolerant

Highly tolerant — LoF variants common in population

Missense Constraint
0.89Z-score
OE missense 0.77 (0.660.92)
95 obs / 122.8 exp
Tolerant

Mild missense constraint

Observed / Expected Ratios
LoF OE0.66 (0.361.30)
00.351.4
Missense OE0.77 (0.660.92)
00.61.4
Synonymous OE0.88
01.21.6
LoF obs/exp: 6 / 9.1Missense obs/exp: 95 / 122.8Syn Z: 0.63
DN
0.79top 25%
GOF
0.77top 25%
LOF
0.2873th %ile

This gene has evidence for multiple mechanisms of pathogenicity (dominant-negative and gain-of-function). Both the Badonyi & Marsh prediction and the broader genomic evidence point to dominant-negative as the predominant mechanism. Different variants in this gene may act through different mechanisms — interpret in context of the specific variant.

DNprediction above median
GOFprediction above median

Note: In-silico variant effect predictors (SIFT, PolyPhen, REVEL, CADD) may underestimate pathogenicity of missense variants in genes with GOF or DN mechanisms. Consider functional evidence and clinical context.

Predictions from Badonyi M, Marsh JA. PLoS ONE. 2024;19(8):e0307312.

ClinVar Variant Classifications

79 submitted variants in ClinVar

Classification Summary

Pathogenic41
Likely Pathogenic2
VUS26
Likely Benign2
Benign1
41
Pathogenic
2
Likely Pathogenic
26
VUS
2
Likely Benign
1
Benign

Curated Variants Distribution

Classified variants from ClinVar · 5 ACMG categories

ClassificationLoFMissense + InframeNon-codingSynonymousTotal
Pathogenic
0
0
41
0
41
Likely Pathogenic
0
0
2
0
2
VUS
0
17
9
0
26
Likely Benign
0
0
2
0
2
Benign
0
0
1
0
1
Total01755072

LoF = frameshift, stop gained/lost, canonical splice · Counts from ClinVar esearch · Updated hourly

View in ClinVar →

Protein Context — Lollipop Plot

RIT2 · protein map & ClinVar variants

Showing all ClinVar variants across the protein. Search a specific variant to highlight its position.

Clinical Trials

Active and recruiting trials from ClinicalTrials.gov

No active trials found for this gene.

Search ClinicalTrials.gov →
Clinical Literature
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