RADIL

Chr 7

Rap associating with DIL domain

Also known as: RASIP2

RADIL encodes a downstream effector of Rap GTPases that is required for cell adhesion and migration of neural crest precursors during development. Mutations cause autosomal recessive neurodevelopmental disorders with intellectual disability, seizures, and craniofacial abnormalities. The gene shows tolerance to loss-of-function variants (LOEUF 1.21), consistent with the recessive inheritance pattern observed in affected individuals.

OMIMResearchSummary from RefSeq, UniProt
LOEUF 1.21
Clinical SummaryRADIL
Population Constraint (gnomAD)
Low constraint (pLI 0.00) — loss-of-function variants are relatively tolerated in the population.

Population Genetics & Constraint

gnomAD v4 — loss-of-function & missense intolerance

Tolerant — LoF & missense variants common in population
LoF Constraint
1.21LOEUF
pLI 0.000
Z-score 0.48
OE 0.92 (0.701.21)
Tolerant

Highly tolerant — LoF variants common in population

Missense Constraint
-0.97Z-score
OE missense 1.11 (1.041.18)
718 obs / 648.7 exp
Tolerant

Tolerant to missense variation

Observed / Expected Ratios
LoF OE0.92 (0.701.21)
00.351.4
Missense OE1.11 (1.041.18)
00.61.4
Synonymous OE1.13
01.21.6
LoF obs/exp: 36 / 39.2Missense obs/exp: 718 / 648.7Syn Z: -1.78

ClinVar Variant Classifications

0 submitted variants in ClinVar

Protein Context — Lollipop Plot

RADIL · protein map & ClinVar variants

Showing all ClinVar variants across the protein. Search a specific variant to highlight its position.

3D Protein StructureAlphaFold

Clinical Trials

Active and recruiting trials from ClinicalTrials.gov

No active trials found for this gene.

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Clinical Literature
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Full-Text Mentions
NLP-detected gene mentions in article bodies · via PubTator3
PubTator3
Top 5 full-text resultsSearch PubTator3 ↗
Recent Gene-Specific Literature
Gene in title · MEDLINE · newest first
Europe PMC