RABEPK

Chr 9

Rab9 effector protein with kelch motifs

Also known as: RAB9P40, bA65N13.1, p40

The protein functions as a Rab9 effector required for endosome to trans-Golgi network transport and is predicted to be involved in receptor-mediated endocytosis and vesicle docking during exocytosis. Based on the extremely low pLI score (2.8x10⁻¹¹) and elevated LOEUF score (1.392), this gene appears highly tolerant to loss-of-function mutations, suggesting pathogenic variants may not cause severe disease. No specific diseases or inheritance patterns have been established for RABEPK mutations.

OMIMResearchSummary from RefSeq, UniProt
LOEUF 1.39
Clinical SummaryRABEPK
Population Constraint (gnomAD)
Low constraint (pLI 0.00) — loss-of-function variants are relatively tolerated in the population.

Population Genetics & Constraint

gnomAD v4 — loss-of-function & missense intolerance

Tolerant — LoF & missense variants common in population
LoF Constraint
1.39LOEUF
pLI 0.000
Z-score 0.28
OE 0.93 (0.631.39)
Tolerant

Highly tolerant — LoF variants common in population

Missense Constraint
0.05Z-score
OE missense 0.99 (0.881.11)
207 obs / 208.9 exp
Tolerant

Mild missense constraint

Observed / Expected Ratios
LoF OE0.93 (0.631.39)
00.351.4
Missense OE0.99 (0.881.11)
00.61.4
Synonymous OE0.98
01.21.6
LoF obs/exp: 17 / 18.3Missense obs/exp: 207 / 208.9Syn Z: 0.16

ClinVar Variant Classifications

0 submitted variants in ClinVar

Protein Context — Lollipop Plot

RABEPK · protein map & ClinVar variants

Showing all ClinVar variants across the protein. Search a specific variant to highlight its position.

3D Protein StructureAlphaFold

Clinical Trials

Active and recruiting trials from ClinicalTrials.gov

No active trials found for this gene.

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Clinical Literature
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