PRKAB2

Chr 1

protein kinase AMP-activated non-catalytic subunit beta 2

The protein serves as a non-catalytic regulatory subunit of AMP-activated protein kinase (AMPK), an energy sensor that monitors cellular energy status and regulates metabolic pathways by phosphorylating key enzymes involved in fatty acid and cholesterol biosynthesis. Mutations cause Wolff-Parkinson-White syndrome with intellectual disability and other abnormalities, inherited in an autosomal recessive pattern. The gene shows high constraint against loss-of-function variants (LOEUF 0.485), indicating that complete protein loss is poorly tolerated.

GeneReviewsOMIMResearchSummary from RefSeq, UniProt
DNmechanismLOEUF 0.48
Clinical SummaryPRKAB2
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Gene-Disease Validity (ClinGen)
congenital heart disease · UDDisputed

Disputed — evidence questions this relationship

Population Constraint (gnomAD)
Moderately constrained gene (pLI 0.73) — some intolerance to loss-of-function variants.
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ClinVar Variants
285 unique Pathogenic / Likely Pathogenic· 30 VUS of 325 total submissions
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GeneReview available — PRKAB2
Authoritative clinical overview · Recommended first read
Open GeneReview ↗

Population Genetics & Constraint

gnomAD v4 — loss-of-function & missense intolerance

Moderate LoF intolerance
LoF Constraint
0.48LOEUF
pLI 0.726
Z-score 2.82
OE 0.15 (0.060.48)
Moderately constrained

More LoF-intolerant than ~75% of genes

Missense Constraint
1.50Z-score
OE missense 0.65 (0.550.77)
97 obs / 148.4 exp
Tolerant

Mild missense constraint

Observed / Expected Ratios
LoF OE0.15 (0.060.48)
00.351.4
Missense OE0.65 (0.550.77)
00.61.4
Synonymous OE0.94
01.21.6
LoF obs/exp: 2 / 13.0Missense obs/exp: 97 / 148.4Syn Z: 0.38
DN
0.6356th %ile
GOF
0.3986th %ile
LOF
0.58top 25%

The highest-scoring mechanism for this gene is dominant-negative.

DNprediction above median

Note: In-silico variant effect predictors (SIFT, PolyPhen, REVEL, CADD) may underestimate pathogenicity of missense variants in genes with GOF or DN mechanisms. Consider functional evidence and clinical context.

Predictions from Badonyi M, Marsh JA. PLoS ONE. 2024;19(8):e0307312.

ClinVar Variant Classifications

325 submitted variants in ClinVar

Classification Summary

Pathogenic247
Likely Pathogenic38
VUS30
247
Pathogenic
38
Likely Pathogenic
30
VUS

Curated Variants Distribution

Classified variants from ClinVar · 5 ACMG categories· variant type breakdown unavailable

ClassificationLoFMissense + InframeNon-codingSynonymousTotal
Pathogenic
247
Likely Pathogenic
38
VUS
30
Likely Benign
0
Benign
0
Total315

Counts from ClinVar esearch · Updated hourly

View in ClinVar →

Protein Context — Lollipop Plot

PRKAB2 · protein map & ClinVar variants

Showing all ClinVar variants across the protein. Search a specific variant to highlight its position.

3D Protein StructureAlphaFold

Clinical Trials

Active and recruiting trials from ClinicalTrials.gov

No active trials found for this gene.

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Clinical Literature
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