OR11L1

Chr 1

olfactory receptor family 11 subfamily L member 1

This gene encodes an olfactory receptor that detects odorant molecules and initiates G-protein-coupled signaling to trigger smell perception. No disease associations have been established for mutations in OR11L1. The gene shows low constraint against loss-of-function variants, consistent with the redundancy typical of the large olfactory receptor gene family.

ResearchSummary from RefSeq, UniProt
MultiplemechanismLOEUF 1.08
Clinical SummaryOR11L1
Population Constraint (gnomAD)
Low constraint (pLI 0.05) — loss-of-function variants are relatively tolerated in the population.
📋
ClinVar Variants
69 unique Pathogenic / Likely Pathogenic· 64 VUS of 151 total submissions
Some data sources returned errors (1)

omim: Error: OMIM fetch failed: 429

Population Genetics & Constraint

gnomAD v4 — loss-of-function & missense intolerance

Tolerant — LoF & missense variants common in population
LoF Constraint
1.08LOEUF
pLI 0.050
Z-score 1.44
OE 0.42 (0.191.08)
Tolerant

Highly tolerant — LoF variants common in population

Missense Constraint
-0.35Z-score
OE missense 1.07 (0.951.21)
190 obs / 177.0 exp
Tolerant

Tolerant to missense variation

Observed / Expected Ratios
LoF OE0.42 (0.191.08)
00.351.4
Missense OE1.07 (0.951.21)
00.61.4
Synonymous OE1.00
01.21.6
LoF obs/exp: 3 / 7.2Missense obs/exp: 190 / 177.0Syn Z: -0.01
DN
0.88top 5%
GOF
0.82top 10%
LOF
0.1399th %ile

This gene has evidence for multiple mechanisms of pathogenicity (dominant-negative and gain-of-function). Both the Badonyi & Marsh prediction and the broader genomic evidence point to dominant-negative as the predominant mechanism. Different variants in this gene may act through different mechanisms — interpret in context of the specific variant.

DNprediction above median
GOFprediction above median

Note: In-silico variant effect predictors (SIFT, PolyPhen, REVEL, CADD) may underestimate pathogenicity of missense variants in genes with GOF or DN mechanisms. Consider functional evidence and clinical context.

Predictions from Badonyi M, Marsh JA. PLoS ONE. 2024;19(8):e0307312.

ClinVar Variant Classifications

151 submitted variants in ClinVar

Classification Summary

Pathogenic63
Likely Pathogenic6
VUS64
Likely Benign6
Benign12
63
Pathogenic
6
Likely Pathogenic
64
VUS
6
Likely Benign
12
Benign

Curated Variants Distribution

Classified variants from ClinVar · 5 ACMG categories

ClassificationLoFMissense + InframeNon-codingSynonymousTotal
Pathogenic
0
0
63
0
63
Likely Pathogenic
0
0
6
0
6
VUS
0
43
21
0
64
Likely Benign
0
2
3
1
6
Benign
0
0
12
0
12
Total0451051151

LoF = frameshift, stop gained/lost, canonical splice · Counts from ClinVar esearch · Updated hourly

View in ClinVar →

Protein Context — Lollipop Plot

OR11L1 · protein map & ClinVar variants

Showing all ClinVar variants across the protein. Search a specific variant to highlight its position.

3D Protein StructureAlphaFold

Clinical Trials

Active and recruiting trials from ClinicalTrials.gov

No active trials found for this gene.

Search ClinicalTrials.gov →
Clinical Literature
Open Research Assistant →
Key Publications
Landmark & review papers · by relevance
PubMed
Top 1 results · since 2015Search PubMed ↗
Recent Gene-Specific Literature
Gene in title · MEDLINE · newest first
Europe PMC

No open access results found