OR10S1

Chr 11

olfactory receptor family 10 subfamily S member 1

Also known as: OR11-279

OR10S1 encodes an olfactory receptor that detects odorant molecules and initiates neuronal responses for smell perception through G-protein-coupled signaling. This gene shows very low constraint against loss-of-function variants (pLI 0.00005, LOEUF 1.87), and no established disease associations have been reported for OR10S1 mutations. Most olfactory receptor genes are dispensable individually, as the large gene family provides functional redundancy for odor detection.

Summary from RefSeq, UniProt
Research Assistant →
0
Active trials
1
Pubs (1 yr)
54
P/LP submissions
0%
P/LP missense
1.87
LOEUF
Multiple*
Mechanism· predicted
Clinical SummaryOR10S1
Population Constraint (gnomAD)
Low constraint (pLI 0.00) — loss-of-function variants are relatively tolerated in the population.
📋
ClinVar Variants
54 unique Pathogenic / Likely Pathogenic· 61 VUS of 117 total submissions

Population Genetics & Constraint

gnomAD v4 — loss-of-function & missense intolerance

Tolerant — LoF & missense variants common in population
LoF Constraint
1.87LOEUF
pLI 0.000
Z-score -0.37
OE 1.18 (0.631.87)
Tolerant

Highly tolerant — LoF variants common in population

Missense Constraint
-0.40Z-score
OE missense 1.08 (0.971.21)
212 obs / 196.1 exp
Tolerant

Tolerant to missense variation

Observed / Expected Ratios
LoF OE1.18 (0.631.87)
00.351.4
Missense OE1.08 (0.971.21)
00.61.4
Synonymous OE1.15
01.21.6
LoF obs/exp: 6 / 5.1Missense obs/exp: 212 / 196.1Syn Z: -1.05
DN
0.82top 10%
GOF
0.83top 10%
LOF
0.1894th %ile

This gene has evidence for multiple mechanisms of pathogenicity (gain-of-function and dominant-negative). Both the Badonyi & Marsh prediction and the broader genomic evidence point to gain-of-function as the predominant mechanism. Different variants in this gene may act through different mechanisms — interpret in context of the specific variant.

GOFprediction above median
DNprediction above median

Note: In-silico variant effect predictors (SIFT, PolyPhen, REVEL, CADD) may underestimate pathogenicity of missense variants in genes with GOF or DN mechanisms. Consider functional evidence and clinical context.

Predictions from Badonyi M, Marsh JA. PLoS ONE. 2024;19(8):e0307312.

ClinVar Variant Classifications

117 submitted variants in ClinVar

Classification Summary

Pathogenic52
Likely Pathogenic2
VUS61
Likely Benign2
52
Pathogenic
2
Likely Pathogenic
61
VUS
2
Likely Benign

Curated Variants Distribution

Classified variants from ClinVar · 5 ACMG categories

ClassificationLoFMissense + InframeNon-codingSynonymousTotal
Pathogenic
0
0
52
0
52
Likely Pathogenic
0
0
2
0
2
VUS
0
56
5
0
61
Likely Benign
0
2
0
0
2
Benign
0
0
0
0
0
Total058590117

LoF = frameshift, stop gained/lost, canonical splice · Counts from ClinVar esearch · Updated hourly

View in ClinVar →

Protein Context — Lollipop Plot

OR10S1 · protein map & ClinVar variants

Showing all ClinVar variants across the protein. Search a specific variant to highlight its position.

Clinical Trials

Active and recruiting trials from ClinicalTrials.gov

No active trials found for this gene.

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Clinical Literature
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Recent Gene-Specific Literature
Gene in title · MEDLINE · newest first
Europe PMC

No open access results found