NTRK1

Chr 1AR

neurotrophic receptor tyrosine kinase 1

Also known as: MTC, TRK, TRK1, TRKA, Trk-A, p140-TrkA

The protein is a receptor tyrosine kinase that binds nerve growth factor (NGF) and regulates the development, differentiation, and survival of sympathetic and sensory neurons through multiple signaling pathways including MAPK and PI3K-AKT cascades. Mutations cause congenital insensitivity to pain with anhidrosis, a severe autosomal recessive disorder characterized by inability to feel pain, absent sweating, self-injurious behavior, and cognitive disability that manifests from birth. The gene is extremely intolerant to loss-of-function variants (pLI near 1.0), indicating that biallelic mutations are required for disease.

OMIMResearchSummary from RefSeq, OMIM, UniProt
LOFmechanismARLOEUF 0.661 OMIM phenotype
Clinical SummaryNTRK1
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Gene-Disease Validity (ClinGen)
hereditary sensory and autonomic neuropathy type 4 · ARDefinitive

Definitive — sufficient evidence for diagnostic panels

Population Constraint (gnomAD)
Low constraint (pLI 0.00) — loss-of-function variants are relatively tolerated in the population.
📋
ClinVar Variants
53 unique Pathogenic / Likely Pathogenic· 151 VUS of 300 total submissions
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Clinical Trials
12 active or recruiting trials — potential therapeutic options may be available

Population Genetics & Constraint

gnomAD v4 — loss-of-function & missense intolerance

Tolerant — LoF & missense variants common in population
LoF Constraint
0.66LOEUF
pLI 0.000
Z-score 3.19
OE 0.43 (0.290.66)
Tolerant

Typical tolerance to LoF variation

Missense Constraint
1.03Z-score
OE missense 0.87 (0.800.94)
419 obs / 482.6 exp
Tolerant

Mild missense constraint

Observed / Expected Ratios
LoF OE0.43 (0.290.66)
00.351.4
Missense OE0.87 (0.800.94)
00.61.4
Synonymous OE1.00
01.21.6
LoF obs/exp: 16 / 37.0Missense obs/exp: 419 / 482.6Syn Z: 0.04
Curated Mechanism (G2P)Gene2Phenotype (DDG2P) ↗
definitiveNTRK1-related congenital insensitivity to pain with anhidrosisLOFAR

Predictions shown for reference only — model trained on dominant genes, not applicable to AR conditions.

DN
0.78top 25%
GOF
0.85top 5%
LOF
0.2581th %ile

The Badonyi & Marsh prediction model was trained exclusively on dominant disease genes. Predictions are not reliable for genes with autosomal recessive inheritance and are shown at reduced opacity for reference only.

Predictions from Badonyi M, Marsh JA. PLoS ONE. 2024;19(8):e0307312.

ClinVar Variant Classifications

300 submitted variants in ClinVar

Classification Summary

Pathogenic19
Likely Pathogenic34
VUS151
Likely Benign76
Conflicting1
19
Pathogenic
34
Likely Pathogenic
151
VUS
76
Likely Benign
1
Conflicting

Curated Variants Distribution

Classified variants from ClinVar · 5 ACMG categories

ClassificationLoFMissense + InframeNon-codingSynonymousTotal
Pathogenic
13
3
2
1
19
Likely Pathogenic
26
5
2
1
34
VUS
1
144
5
1
151
Likely Benign
0
4
35
37
76
Benign
0
0
0
0
0
Conflicting
1
Total401564440281

LoF = frameshift, stop gained/lost, canonical splice · Counts from ClinVar esearch · Updated hourly

View in ClinVar →

Protein Context — Lollipop Plot

NTRK1 · protein map & ClinVar variants

Showing all ClinVar variants across the protein. Search a specific variant to highlight its position.

3D Protein StructureAlphaFold

Clinical Trials

Active and recruiting trials from ClinicalTrials.gov

Breast CancerCholangiocarcinomaColorectal Cancer

Basket Study of Entrectinib (RXDX-101) for the Treatment of Patients With Solid Tumors Harboring NTRK 1/2/3 (Trk A/B/C), ROS1, or ALK Gene Rearrangements (Fusions)

ACTIVE NOT RECRUITING
NCT02568267Phase PHASE2Hoffmann-La RocheStarted 2015-11-19
Entrectinib
Squamous Cell Tumors of the Head and Neck (HNSCC)

Development of Patient Derived Xenografts (PDXs) and Analysis of Tyrosine Kinase Receptor Expression in Patients With Squamous Cell Head and Neck Cancer (HNSCC) to Study Resistance Mechanisms Induced by Standard Therapy

NOT YET RECRUITING
NCT07471009IRCCS Azienda Ospedaliero-Universitaria di BolognaStarted 2026-03-31
Solid Tumors Harboring NTRK Fusion

A Study to Test the Safety and Efficacy of the Drug Larotrectinib for the Treatment of Tumors With NTRK-fusion in Children

ACTIVE NOT RECRUITING
NCT02637687Phase PHASE1, PHASE2BayerStarted 2015-12-16
Larotrectinib (Vitrakvi, BAY2757556)
Non Small Cell Lung Cancer

A Study to Compare Uliledlimab Combined With Toripalimab, Toripalimab Monotherapy, and Pembrolizumab Monotherapy in Patients With Previously Untreated Locally Advanced Unresectable or Metastatic PD-L1- and CD73- Selected Non-Small Cell Lung Cancer

ACTIVE NOT RECRUITING
NCT06984588Phase PHASE2, PHASE3TJ Biopharma Co., Ltd.Started 2024-03-28
UliledlimabToripalimabPembrolizumab
Breast NeoplasmBreast CancerBreast Tumors

Fudan University Shanghai Cancer Center Breast Cancer Precision Platform Series Study- Neoadjuvant Therapy

RECRUITING
NCT05582499Phase PHASE2Fudan UniversityStarted 2022-11-01
DalpiciclibPyrotinibSHR-A1811
NTRK1/2/3 Fusion Gene Detection

NTRK 1,2,3 Rearrangements in Patients With Solid Tumors

ACTIVE NOT RECRUITING
NCT06780384Federico CappuzzoStarted 2020-02-05
Locally Advanced Solid TumorsMetastatic Solid Tumors

A Study of Repotrectinib (TPX-0005) in Patients With Advanced Solid Tumors Harboring ALK, ROS1, or NTRK1-3 Rearrangements

RECRUITING
NCT03093116Phase PHASE1, PHASE2Turning Point Therapeutics, Inc.Started 2017-03-07
Oral repotrectinib (TPX-0005)
Differentiated Thyroid CarcinomaMedullary Thyroid Carcinoma

Spanish Study for Molecular Characterization of Thyroid Carcinoma

ACTIVE NOT RECRUITING
NCT04970134Grupo Español de Tratamiento de Tumores de Cabeza y CuelloStarted 2021-06-14
Immunohistochemistry (IHC)Fluorescence In-Situ Hybridization (FISH)Oncomine Focus Assay Platform with 52 genes
Solid TumourHaematological MalignancyMalignant Neoplasm

DETERMINE Trial Treatment Arm 06: Capmatinib in Adult Patients With Cancers Harbouring MET Dysregulations

RECRUITING
NCT06988475Phase PHASE2, PHASE3Cancer Research UKStarted 2024-11-19
Capmatinib
Advanced LymphomaAdvanced Malignant Solid NeoplasmBladder Carcinoma

Targeted Therapy Directed by Genetic Testing in Treating Patients With Advanced Refractory Solid Tumors, Lymphomas, or Multiple Myeloma (The MATCH Screening Trial)

ACTIVE NOT RECRUITING
NCT02465060Phase PHASE2National Cancer Institute (NCI)Started 2015-08-17
AdavosertibAfatinibAfatinib Dimaleate
Advanced Malignant Solid NeoplasmAnn Arbor Stage III Non-Hodgkin LymphomaAnn Arbor Stage IV Non-Hodgkin Lymphoma

Targeted Therapy Directed by Genetic Testing in Treating Pediatric Patients With Relapsed or Refractory Advanced Solid Tumors, Non-Hodgkin Lymphomas, or Histiocytic Disorders (The Pediatric MATCH Screening Trial)

ACTIVE NOT RECRUITING
NCT03155620Phase PHASE2National Cancer Institute (NCI)Started 2017-07-31
Biopsy ProcedureBiospecimen CollectionBone Marrow Aspiration and Biopsy
Haematological MalignancySolid Tumour

DETERMINE (Determining Extended Therapeutic Indications for Existing Drugs in Rare Molecularly Defined Indications Using a National Evaluation Platform Trial) - Master Screening Protocol

RECRUITING
NCT05722886Phase PHASE2, PHASE3Cancer Research UKStarted 2023-03-01
AlectinibAtezolizumabEntrectinib
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