NDUFB7

Chr 19AR

NADH:ubiquinone oxidoreductase subunit B7

Also known as: B18, CI-B18, MC1DN39

The protein is an accessory subunit of mitochondrial complex I (NADH dehydrogenase), which transfers electrons from NADH to ubiquinone in the respiratory chain. Mutations cause mitochondrial complex I deficiency with autosomal recessive inheritance, though the clinical phenotype remains incompletely characterized. The gene shows low constraint to loss-of-function variants, which is consistent with recessive inheritance patterns.

Summary from RefSeq, OMIM, UniProt
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Primary Disease Associations & Inheritance

?Mitochondrial complex I deficiency, nuclear type 39MIM #620135
AR
0
Active trials
9
Pubs (1 yr)
16
P/LP submissions
0%
P/LP missense
1.67
LOEUF
LOF
Mechanism· G2P
Clinical SummaryNDUFB7
🧬
Gene-Disease Validity (ClinGen)
mitochondrial disease · ARLimited

Limited evidence — not for standalone diagnostic reporting

Population Constraint (gnomAD)
Low constraint (pLI 0.00) — loss-of-function variants are relatively tolerated in the population.
📋
ClinVar Variants
15 unique Pathogenic / Likely Pathogenic· 25 VUS of 50 total submissions

Population Genetics & Constraint

gnomAD v4 — loss-of-function & missense intolerance

Tolerant — LoF & missense variants common in population
LoF Constraint
1.67LOEUF
pLI 0.003
Z-score 0.45
OE 0.78 (0.381.67)
Tolerant

Highly tolerant — LoF variants common in population

Missense Constraint
0.27Z-score
OE missense 0.92 (0.771.10)
86 obs / 93.3 exp
Tolerant

Mild missense constraint

Observed / Expected Ratios
LoF OE0.78 (0.381.67)
00.351.4
Missense OE0.92 (0.771.10)
00.61.4
Synonymous OE0.97
01.21.6
LoF obs/exp: 4 / 5.1Missense obs/exp: 86 / 93.3Syn Z: 0.14
Curated Mechanism (G2P)Gene2Phenotype (DDG2P) ↗
limitedNDUFB7-related lactic acidosis and hypertrophic cardiomyopathyLOFAR

Predictions shown for reference only — model trained on dominant genes, not applicable to AR conditions.

DN
0.6551th %ile
GOF
0.6248th %ile
LOF
0.2091th %ile

The Badonyi & Marsh prediction model was trained exclusively on dominant disease genes. Predictions are not reliable for genes with autosomal recessive inheritance and are shown at reduced opacity for reference only.

Predictions from Badonyi M, Marsh JA. PLoS ONE. 2024;19(8):e0307312.

ClinVar Variant Classifications

50 submitted variants in ClinVar

Classification Summary

Pathogenic13
Likely Pathogenic2
VUS25
Likely Benign3
13
Pathogenic
2
Likely Pathogenic
25
VUS
3
Likely Benign

Curated Variants Distribution

Classified variants from ClinVar · 5 ACMG categories

ClassificationLoFMissense + InframeNon-codingSynonymousTotal
Pathogenic
0
0
13
0
13
Likely Pathogenic
0
0
2
0
2
VUS
0
21
4
0
25
Likely Benign
0
2
1
0
3
Benign
0
0
0
0
0
Total02320043

LoF = frameshift, stop gained/lost, canonical splice · Counts from ClinVar esearch · Updated hourly

View in ClinVar →

Protein Context — Lollipop Plot

NDUFB7 · protein map & ClinVar variants

Showing all ClinVar variants across the protein. Search a specific variant to highlight its position.

Clinical Trials

Active and recruiting trials from ClinicalTrials.gov

No active trials found for this gene.

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Clinical Literature
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