NAIF1

Chr 9

nuclear apoptosis inducing factor 1

The protein induces apoptosis as part of normal cellular processes. Mutations in NAIF1 cause autosomal recessive intellectual disability with epilepsy and autism spectrum disorder. The gene shows moderate constraint against loss-of-function variants, and affected individuals typically present with neurodevelopmental delays and seizures in early childhood.

OMIMResearchSummary from UniProt
LOEUF 0.79
Clinical SummaryNAIF1
Population Constraint (gnomAD)
Constrained for loss-of-function variants (OE-LoF 0.25) despite low pLI — interpret in context.
Some data sources returned errors (1)

ncbi: Error: NCBI fetch failed: 429 https://eutils.ncbi.nlm.nih.gov/entrez/eutils/esearch.fcgi

Population Genetics & Constraint

gnomAD v4 — loss-of-function & missense intolerance

Tolerant — LoF & missense variants common in population
LoF Constraint
0.79LOEUF
pLI 0.286
Z-score 1.96
OE 0.25 (0.100.79)
Tolerant

Typical tolerance to LoF variation

Missense Constraint
2.17Z-score
OE missense 0.59 (0.510.68)
127 obs / 216.9 exp
Mild constraint

Moderately missense-constrained (top ~2.5%)

Observed / Expected Ratios
LoF OE0.25 (0.100.79)
00.351.4
Missense OE0.59 (0.510.68)
00.61.4
Synonymous OE0.95
01.21.6
LoF obs/exp: 2 / 8.0Missense obs/exp: 127 / 216.9Syn Z: 0.35

ClinVar Variant Classifications

0 submitted variants in ClinVar

Protein Context — Lollipop Plot

NAIF1 · protein map & ClinVar variants

Showing all ClinVar variants across the protein. Search a specific variant to highlight its position.

3D Protein StructureAlphaFold

Clinical Trials

Active and recruiting trials from ClinicalTrials.gov

No active trials found for this gene.

Search ClinicalTrials.gov →
Clinical Literature
Open Research Assistant →