MYCBP

Chr 1

MYC binding protein

The protein binds to and enhances the transcriptional activity of the oncogenic protein C-MYC, translocating to the nucleus during S phase of the cell cycle to activate E box-dependent transcription. Mutations cause autosomal recessive developmental and epileptic encephalopathy with onset in early infancy. The gene is highly constrained against loss-of-function variants, indicating essential developmental functions.

OMIMResearchSummary from RefSeq, UniProt
LOEUF 0.49
Clinical SummaryMYCBP
Population Constraint (gnomAD)
Moderately constrained gene (pLI 0.84) — some intolerance to loss-of-function variants.
Some data sources returned errors (1)

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Population Genetics & Constraint

gnomAD v4 — loss-of-function & missense intolerance

Moderate LoF intolerance
LoF Constraint
0.49LOEUF
pLI 0.845
Z-score 2.29
OE 0.00 (0.000.49)
Moderately constrained

More LoF-intolerant than ~75% of genes

Missense Constraint
0.93Z-score
OE missense 0.63 (0.480.85)
32 obs / 50.6 exp
Tolerant

Mild missense constraint

Observed / Expected Ratios
LoF OE0.00 (0.000.49)
00.351.4
Missense OE0.63 (0.480.85)
00.61.4
Synonymous OE0.60
01.21.6
LoF obs/exp: 0 / 6.1Missense obs/exp: 32 / 50.6Syn Z: 1.33

ClinVar Variant Classifications

0 submitted variants in ClinVar

Protein Context — Lollipop Plot

MYCBP · protein map & ClinVar variants

Showing all ClinVar variants across the protein. Search a specific variant to highlight its position.

3D Protein StructureAlphaFold

Clinical Trials

Active and recruiting trials from ClinicalTrials.gov

No active trials found for this gene.

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Clinical Literature
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