MPPE1

Chr 18

metallophosphoesterase 1

Also known as: Cdc1, PGAP5

The protein is a metallophosphoesterase that removes ethanolamine-phosphate from GPI-anchor protein intermediates during glycan remodeling, enabling efficient transport of GPI-anchored proteins from the endoplasmic reticulum to the Golgi apparatus. Mutations in MPPE1 cause congenital disorder of glycosylation type Iq, which presents with intellectual disability, seizures, and hypotonia with autosomal recessive inheritance. The gene shows relaxed evolutionary constraint with a high LOEUF score of 1.207, indicating tolerance to loss-of-function variants in the general population.

Summary from RefSeq, UniProt
Research Assistant →
1
Active trials
2
Pubs (1 yr)
95
P/LP submissions
0%
P/LP missense
1.21
LOEUF
DN
Mechanism· predicted
Clinical SummaryMPPE1
Population Constraint (gnomAD)
Low constraint (pLI 0.00) — loss-of-function variants are relatively tolerated in the population.
📋
ClinVar Variants
91 unique Pathogenic / Likely Pathogenic· 83 VUS of 194 total submissions
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Clinical Trials
1 active or recruiting trial — potential therapeutic options may be available

Population Genetics & Constraint

gnomAD v4 — loss-of-function & missense intolerance

Tolerant — LoF & missense variants common in population
LoF Constraint
1.21LOEUF
pLI 0.000
Z-score 0.85
OE 0.79 (0.541.21)
Tolerant

Highly tolerant — LoF variants common in population

Missense Constraint
0.35Z-score
OE missense 0.93 (0.831.05)
210 obs / 224.9 exp
Tolerant

Mild missense constraint

Observed / Expected Ratios
LoF OE0.79 (0.541.21)
00.351.4
Missense OE0.93 (0.831.05)
00.61.4
Synonymous OE1.01
01.21.6
LoF obs/exp: 16 / 20.1Missense obs/exp: 210 / 224.9Syn Z: -0.05
DN
0.6161th %ile
GOF
0.5759th %ile
LOF
0.3260th %ile

The highest-scoring mechanism for this gene is dominant-negative.

DNprediction above median

Note: In-silico variant effect predictors (SIFT, PolyPhen, REVEL, CADD) may underestimate pathogenicity of missense variants in genes with GOF or DN mechanisms. Consider functional evidence and clinical context.

Predictions from Badonyi M, Marsh JA. PLoS ONE. 2024;19(8):e0307312.

ClinVar Variant Classifications

194 submitted variants in ClinVar

Classification Summary

Pathogenic89
Likely Pathogenic2
VUS83
Likely Benign6
89
Pathogenic
2
Likely Pathogenic
83
VUS
6
Likely Benign

Curated Variants Distribution

Classified variants from ClinVar · 5 ACMG categories

ClassificationLoFMissense + InframeNon-codingSynonymousTotal
Pathogenic
0
0
89
0
89
Likely Pathogenic
0
0
2
0
2
VUS
0
68
15
0
83
Likely Benign
0
6
0
0
6
Benign
0
0
0
0
0
Total0741060180

LoF = frameshift, stop gained/lost, canonical splice · Counts from ClinVar esearch · Updated hourly

View in ClinVar →

Protein Context — Lollipop Plot

MPPE1 · protein map & ClinVar variants

Showing all ClinVar variants across the protein. Search a specific variant to highlight its position.

Clinical Literature
Open Research Assistant →
Recent Gene-Specific Literature
Gene in title · MEDLINE · newest first
Europe PMC