MED30

Chr 8

mediator complex subunit 30

Also known as: MED30S, THRAP6, TRAP25

MED30 encodes a component of the Mediator complex, which serves as a bridge between gene-specific regulatory proteins and RNA polymerase II to facilitate transcription of nearly all protein-coding genes. Mutations cause autosomal recessive intellectual disability with seizures and spastic paraplegia, typically presenting in early childhood. The gene shows moderate constraint against loss-of-function variants (LOEUF 0.67), consistent with its essential role in transcriptional regulation.

OMIMResearchSummary from RefSeq, UniProt
LOEUF 0.67
Clinical SummaryMED30
Population Constraint (gnomAD)
Constrained for loss-of-function variants (OE-LoF 0.21) despite low pLI — interpret in context.

Population Genetics & Constraint

gnomAD v4 — loss-of-function & missense intolerance

Tolerant — LoF & missense variants common in population
LoF Constraint
0.67LOEUF
pLI 0.403
Z-score 2.23
OE 0.21 (0.090.67)
Tolerant

Typical tolerance to LoF variation

Missense Constraint
0.69Z-score
OE missense 0.80 (0.670.97)
78 obs / 97.2 exp
Tolerant

Mild missense constraint

Observed / Expected Ratios
LoF OE0.21 (0.090.67)
00.351.4
Missense OE0.80 (0.670.97)
00.61.4
Synonymous OE1.00
01.21.6
LoF obs/exp: 2 / 9.4Missense obs/exp: 78 / 97.2Syn Z: -0.01

ClinVar Variant Classifications

0 submitted variants in ClinVar

Protein Context — Lollipop Plot

MED30 · protein map & ClinVar variants

Showing all ClinVar variants across the protein. Search a specific variant to highlight its position.

3D Protein StructureAlphaFold

Clinical Trials

Active and recruiting trials from ClinicalTrials.gov

No active trials found for this gene.

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Clinical Literature
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