MBD5

Chr 2AD

methyl-CpG binding domain protein 5

Also known as: C2DELq23.1, DEL2Q23.1, MRD1

The protein binds specifically to methylated DNA and interacts with the polycomb repressive complex PR-DUB to regulate histone deubiquitination. Haploinsufficiency causes autosomal dominant intellectual developmental disorder characterized by microcephaly, intellectual disabilities, severe speech impairment, and seizures. The pathogenic mechanism involves loss of function mutations leading to reduced gene expression.

Summary from RefSeq, OMIM, UniProt, Mechanism
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Primary Disease Associations & Inheritance

Intellectual developmental disorder, autosomal dominant 1MIM #156200
AD
2
Active trials
10
Pubs (1 yr)
31
P/LP submissions
3%
P/LP missense
0.14
LOEUF· LoF intol.
LOF
Mechanism· G2P
Clinical SummaryMBD5
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Gene-Disease Validity (ClinGen)
complex neurodevelopmental disorder · ADDefinitive

Definitive — sufficient evidence for diagnostic panels

Population Constraint (gnomAD)
Highly constrained gene — heterozygous loss-of-function variants are very rare in the population (pLI 1.00). One damaged copy is likely sufficient to cause disease.
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ClinVar Variants
31 unique Pathogenic / Likely Pathogenic· 340 VUS of 500 total submissions
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Clinical Trials
2 active or recruiting trials — potential therapeutic options may be available
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GeneReview available — MBD5
Authoritative clinical overview · Recommended first read
Open GeneReview ↗

Population Genetics & Constraint

gnomAD v4 — loss-of-function & missense intolerance

LoF intolerant — likely haploinsufficient
LoF Constraint
0.14LOEUF
pLI 1.000
Z-score 6.04
OE 0.04 (0.020.14)
Highly constrained

Among the most LoF-intolerant genes (~top 3%)

Missense Constraint
0.86Z-score
OE missense 0.91 (0.860.97)
710 obs / 777.1 exp
Tolerant

Mild missense constraint

Observed / Expected Ratios
LoF OE0.04 (0.020.14)
00.351.4
Missense OE0.91 (0.860.97)
00.61.4
Synonymous OE1.05
01.21.6
LoF obs/exp: 2 / 46.4Missense obs/exp: 710 / 777.1Syn Z: -0.61
Curated Mechanism (G2P)Gene2Phenotype (DDG2P) ↗
strongMBD5-related intellectual developmental disorderLOFAD
DN
0.2897th %ile
GOF
0.1999th %ile
LOF
0.85top 5%

The highest-scoring mechanism for this gene is loss-of-function (haploinsufficiency).

LOFprediction above median · 1 literature citation · 52% of P/LP variants are LoF · LOEUF 0.14

Literature Evidence

LOFMBD5 haploinsufficiency is a neurodevelopmental disorder characterized by developmental delay / intellectual disability, severe speech impairment, seizures, sleep disturbances, and abnormal behaviors.PMID:27786435

Predictions from Badonyi M, Marsh JA. PLoS ONE. 2024;19(8):e0307312. Mechanism ranking also informed by gnomAD constraint, ClinVar, and ClinGen data.

ClinVar Variant Classifications

500 submitted variants in ClinVar

Classification Summary

Pathogenic17
Likely Pathogenic14
VUS340
Likely Benign105
Benign1
Conflicting7
17
Pathogenic
14
Likely Pathogenic
340
VUS
105
Likely Benign
1
Benign
7
Conflicting

Curated Variants Distribution

Classified variants from ClinVar · 5 ACMG categories

ClassificationLoFMissense + InframeNon-codingSynonymousTotal
Pathogenic
11
0
6
0
17
Likely Pathogenic
5
1
8
0
14
VUS
1
324
12
3
340
Likely Benign
0
7
22
76
105
Benign
0
0
1
0
1
Conflicting
7
Total173324979484

LoF = frameshift, stop gained/lost, canonical splice · Counts from ClinVar esearch · Updated hourly

View in ClinVar →

Protein Context — Lollipop Plot

MBD5 · protein map & ClinVar variants

Showing all ClinVar variants across the protein. Search a specific variant to highlight its position.

Clinical Literature
Open Research Assistant →
Full-Text Mentions
NLP-detected gene mentions in article bodies · via PubTator3
PubTator3
Top 5 full-text resultsSearch PubTator3 ↗