MAP3K7

Chr 6AD

mitogen-activated protein kinase kinase kinase 7

Also known as: CSCF, FMD2, MEKK7, TAK1, TGF1a

MAP3K7 encodes a serine/threonine kinase that mediates signal transduction for multiple pathways including TGF-beta, BMP, and cytokine signaling, and serves as a key activator of NF-kappa-B and MAPK pathways in response to environmental stresses. Mutations cause autosomal dominant developmental delay, intellectual disability, and multiple congenital anomalies, often with cardiac defects and distinctive facial features. This gene is highly constrained against loss-of-function variants (pLI = 1.0, LOEUF = 0.21), reflecting its essential role in cellular signaling pathways.

GeneReviewsOMIMResearchSummary from RefSeq, UniProt
LOFmechanismADLOEUF 0.212 OMIM phenotypes
Clinical SummaryMAP3K7
Population Constraint (gnomAD)
Highly constrained gene — heterozygous loss-of-function variants are very rare in the population (pLI 1.00). One damaged copy is likely sufficient to cause disease.
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ClinVar Variants
28 unique Pathogenic / Likely Pathogenic· 120 VUS of 234 total submissions
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GeneReview available — MAP3K7
Authoritative clinical overview · Recommended first read
Open GeneReview ↗

Population Genetics & Constraint

gnomAD v4 — loss-of-function & missense intolerance

LoF intolerant — likely haploinsufficient
LoF Constraint
0.21LOEUF
pLI 1.000
Z-score 5.14
OE 0.08 (0.040.21)
Highly constrained

Highly LoF-intolerant (top ~10% of genes)

Missense Constraint
2.79Z-score
OE missense 0.57 (0.510.65)
195 obs / 339.4 exp
Mild constraint

Moderately missense-constrained (top ~2.5%)

Observed / Expected Ratios
LoF OE0.08 (0.040.21)
00.351.4
Missense OE0.57 (0.510.65)
00.61.4
Synonymous OE0.97
01.21.6
LoF obs/exp: 3 / 36.5Missense obs/exp: 195 / 339.4Syn Z: 0.26
Curated Mechanism (G2P)Gene2Phenotype (DDG2P) ↗
strongMAP3K7-related frontometaphyseal dysplasiaOTHERAD
DN
0.4190th %ile
GOF
0.5759th %ile
LOF
0.74top 10%

The highest-scoring mechanism for this gene is loss-of-function (haploinsufficiency).

LOFprediction above median · 1 literature citation · LOEUF 0.21

Literature Evidence

LOFTogether with the demonstrated tumor suppressive function in cell line experiments and lacking evidence for inactivation through hypermethylation, these results indicate MAP3K7 as a gene for which haploinsufficency is substantially tumorigenic.PMID:23370768

Predictions from Badonyi M, Marsh JA. PLoS ONE. 2024;19(8):e0307312. Mechanism ranking also informed by gnomAD constraint, ClinVar, and ClinGen data.

ClinVar Variant Classifications

234 submitted variants in ClinVar

Classification Summary

Pathogenic20
Likely Pathogenic8
VUS120
Likely Benign70
Benign7
Conflicting9
20
Pathogenic
8
Likely Pathogenic
120
VUS
70
Likely Benign
7
Benign
9
Conflicting

Curated Variants Distribution

Classified variants from ClinVar · 5 ACMG categories

ClassificationLoFMissense + InframeNon-codingSynonymousTotal
Pathogenic
0
9
11
0
20
Likely Pathogenic
0
8
0
0
8
VUS
8
86
22
4
120
Likely Benign
0
5
41
24
70
Benign
0
1
5
1
7
Conflicting
9
Total81097929234

LoF = frameshift, stop gained/lost, canonical splice · Counts from ClinVar esearch · Updated hourly

View in ClinVar →

Protein Context — Lollipop Plot

MAP3K7 · protein map & ClinVar variants

Showing all ClinVar variants across the protein. Search a specific variant to highlight its position.

3D Protein StructureAlphaFold

Clinical Trials

Active and recruiting trials from ClinicalTrials.gov

No active trials found for this gene.

Search ClinicalTrials.gov →
Clinical Literature
Open Research Assistant →
Full-Text Mentions
NLP-detected gene mentions in article bodies · via PubTator3
PubTator3
Top 5 full-text resultsSearch PubTator3 ↗
Key Publications
Landmark & review papers · by relevance
PubMed
Top 5 results · since 2015Search PubMed ↗