LYPD6 encodes a protein that modulates nicotinic acetylcholine receptor function in the brain, specifically inhibiting alpha-3:beta-4 and alpha-7 receptor currents in an allosteric manner. Mutations cause autosomal recessive intellectual disability with seizures and language delay. The gene shows low constraint against loss-of-function variants, consistent with a recessive inheritance pattern.

OMIMResearchSummary from RefSeq, UniProt
LOEUF 1.48
Clinical SummaryLYPD6
Population Constraint (gnomAD)
Low constraint (pLI 0.00) — loss-of-function variants are relatively tolerated in the population.

Population Genetics & Constraint

gnomAD v4 — loss-of-function & missense intolerance

Tolerant — LoF & missense variants common in population
LoF Constraint
1.48LOEUF
pLI 0.000
Z-score 0.44
OE 0.85 (0.511.48)
Tolerant

Highly tolerant — LoF variants common in population

Missense Constraint
0.33Z-score
OE missense 0.91 (0.761.08)
87 obs / 96.1 exp
Tolerant

Mild missense constraint

Observed / Expected Ratios
LoF OE0.85 (0.511.48)
00.351.4
Missense OE0.91 (0.761.08)
00.61.4
Synonymous OE0.93
01.21.6
LoF obs/exp: 9 / 10.5Missense obs/exp: 87 / 96.1Syn Z: 0.33

ClinVar Variant Classifications

0 submitted variants in ClinVar

Protein Context — Lollipop Plot

LYPD6 · protein map & ClinVar variants

Showing all ClinVar variants across the protein. Search a specific variant to highlight its position.

3D Protein StructureAlphaFold

Clinical Trials

Active and recruiting trials from ClinicalTrials.gov

No active trials found for this gene.

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Clinical Literature
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