The protein binds galactosides and is structurally highly similar to galectin 9. This gene shows low constraint against loss-of-function variants (pLI < 0.1, LOEUF > 1), suggesting tolerance to protein loss. No established disease associations have been reported for mutations in this gene.

ResearchSummary from RefSeq, UniProt
DNmechanismLOEUF 1.17
Clinical SummaryLGALS9B
Population Constraint (gnomAD)
Low constraint (pLI 0.00) — loss-of-function variants are relatively tolerated in the population.

Population Genetics & Constraint

gnomAD v4 — loss-of-function & missense intolerance

Tolerant — LoF & missense variants common in population
LoF Constraint
1.17LOEUF
pLI 0.000
Z-score 1.15
OE 0.65 (0.381.17)
Tolerant

Highly tolerant — LoF variants common in population

Missense Constraint
0.54Z-score
OE missense 0.88 (0.781.01)
154 obs / 174.1 exp
Tolerant

Mild missense constraint

Observed / Expected Ratios
LoF OE0.65 (0.381.17)
00.351.4
Missense OE0.88 (0.781.01)
00.61.4
Synonymous OE0.93
01.21.6
LoF obs/exp: 8 / 12.4Missense obs/exp: 154 / 174.1Syn Z: 0.44
DN
0.76top 25%
GOF
0.4381th %ile
LOF
0.3065th %ile

The highest-scoring mechanism for this gene is dominant-negative.

DNprediction above median

Note: In-silico variant effect predictors (SIFT, PolyPhen, REVEL, CADD) may underestimate pathogenicity of missense variants in genes with GOF or DN mechanisms. Consider functional evidence and clinical context.

Predictions from Badonyi M, Marsh JA. PLoS ONE. 2024;19(8):e0307312.

ClinVar Variant Classifications

0 submitted variants in ClinVar

Protein Context — Lollipop Plot

LGALS9B · protein map & ClinVar variants

Showing all ClinVar variants across the protein. Search a specific variant to highlight its position.

3D Protein StructureAlphaFold

Clinical Trials

Active and recruiting trials from ClinicalTrials.gov

No active trials found for this gene.

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