ISG15

Chr 1AR

ISG15 ubiquitin like modifier

Also known as: G1P2, IFI15, IMD38, IP17, UCRP, hUCRP

The encoded protein is a ubiquitin-like modifier that conjugates to target proteins (ISGylation) and functions as a secreted cytokine, both critical for antiviral immunity and interferon signaling. Mutations cause immunodeficiency 38, an autosomal recessive primary immunodeficiency characterized by increased susceptibility to viral infections and mycobacterial disease. The gene shows moderate tolerance to loss-of-function variants (LOEUF 1.691), consistent with the recessive inheritance pattern where biallelic mutations are required for disease.

Summary from RefSeq, OMIM, UniProt
Research Assistant →

Primary Disease Associations & Inheritance

Immunodeficiency 38MIM #616126
AR
2
Active trials
316
Pubs (1 yr)
135
P/LP submissions
0%
P/LP missense
1.69
LOEUF
DN
Mechanism· predicted
Clinical SummaryISG15
Population Constraint (gnomAD)
Constrained for loss-of-function variants (OE-LoF 0.00) despite low pLI — interpret in context.
📋
ClinVar Variants
126 unique Pathogenic / Likely Pathogenic· 92 VUS of 304 total submissions
💊
Clinical Trials
2 active or recruiting trials — potential therapeutic options may be available

Population Genetics & Constraint

gnomAD v4 — loss-of-function & missense intolerance

Tolerant — LoF & missense variants common in population
LoF Constraint
1.69LOEUF
pLI 0.405
Z-score 0.97
OE 0.00 (0.001.69)
Tolerant

Highly tolerant — LoF variants common in population

Missense Constraint
-0.04Z-score
OE missense 1.01 (0.871.18)
117 obs / 115.7 exp
Tolerant

Tolerant to missense variation

Observed / Expected Ratios
LoF OE0.00 (0.001.69)
00.351.4
Missense OE1.01 (0.871.18)
00.61.4
Synonymous OE1.08
01.21.6
LoF obs/exp: 0 / 1.1Missense obs/exp: 117 / 115.7Syn Z: -0.50
DN
0.6552th %ile
GOF
0.5856th %ile
LOF
0.3939th %ile

The highest-scoring mechanism for this gene is dominant-negative.

DNprediction above median

Note: In-silico variant effect predictors (SIFT, PolyPhen, REVEL, CADD) may underestimate pathogenicity of missense variants in genes with GOF or DN mechanisms. Consider functional evidence and clinical context.

Predictions from Badonyi M, Marsh JA. PLoS ONE. 2024;19(8):e0307312.

ClinVar Variant Classifications

304 submitted variants in ClinVar

Classification Summary

Pathogenic119
Likely Pathogenic7
VUS92
Likely Benign64
Benign15
Conflicting6
119
Pathogenic
7
Likely Pathogenic
92
VUS
64
Likely Benign
15
Benign
6
Conflicting

Curated Variants Distribution

Classified variants from ClinVar · 5 ACMG categories

ClassificationLoFMissense + InframeNon-codingSynonymousTotal
Pathogenic
1
0
118
0
119
Likely Pathogenic
1
0
6
0
7
VUS
5
62
25
0
92
Likely Benign
0
3
11
50
64
Benign
0
1
8
6
15
Conflicting
6
Total76616856303

LoF = frameshift, stop gained/lost, canonical splice · Counts from ClinVar esearch · Updated hourly

View in ClinVar →

Protein Context — Lollipop Plot

ISG15 · protein map & ClinVar variants

Showing all ClinVar variants across the protein. Search a specific variant to highlight its position.

Clinical Literature
Open Research Assistant →
Full-Text Mentions
NLP-detected gene mentions in article bodies · via PubTator3
PubTator3
Top 5 full-text resultsSearch PubTator3 ↗