GRK6

Chr 5

G protein-coupled receptor kinase 6

Also known as: GPRK6

The protein phosphorylates activated G protein-coupled receptors to initiate their desensitization and internalization, with particular involvement in dopamine receptor regulation in the striatum and chemokine receptor signaling. Mutations cause autosomal recessive dystonia-26 with early childhood onset, characterized by progressive movement disorders affecting the limbs and sometimes involving additional neurological features. The gene is highly constrained against loss-of-function variants, indicating that complete loss of protein function is poorly tolerated.

Summary from RefSeq, UniProt
Research Assistant →
0
Active trials
11
Pubs (1 yr)
57
P/LP submissions
0%
P/LP missense
0.57
LOEUF
GOF
Mechanism· predicted
Clinical SummaryGRK6
Population Constraint (gnomAD)
Low constraint (pLI 0.00) — loss-of-function variants are relatively tolerated in the population.
📋
ClinVar Variants
55 unique Pathogenic / Likely Pathogenic· 65 VUS of 141 total submissions

Population Genetics & Constraint

gnomAD v4 — loss-of-function & missense intolerance

Moderate LoF intolerance
LoF Constraint
0.57LOEUF
pLI 0.000
Z-score 3.50
OE 0.35 (0.230.57)
Moderately constrained

More LoF-intolerant than ~75% of genes

Missense Constraint
2.81Z-score
OE missense 0.60 (0.540.67)
234 obs / 390.2 exp
Mild constraint

Moderately missense-constrained (top ~2.5%)

Observed / Expected Ratios
LoF OE0.35 (0.230.57)
00.351.4
Missense OE0.60 (0.540.67)
00.61.4
Synonymous OE0.98
01.21.6
LoF obs/exp: 12 / 34.1Missense obs/exp: 234 / 390.2Syn Z: 0.17
DN
0.5772th %ile
GOF
0.6736th %ile
LOF
0.3843th %ile

The highest-scoring mechanism for this gene is gain-of-function.

GOFprediction above median

Note: In-silico variant effect predictors (SIFT, PolyPhen, REVEL, CADD) may underestimate pathogenicity of missense variants in genes with GOF or DN mechanisms. Consider functional evidence and clinical context.

Predictions from Badonyi M, Marsh JA. PLoS ONE. 2024;19(8):e0307312.

ClinVar Variant Classifications

141 submitted variants in ClinVar

Classification Summary

Pathogenic50
Likely Pathogenic5
VUS65
Likely Benign5
50
Pathogenic
5
Likely Pathogenic
65
VUS
5
Likely Benign

Curated Variants Distribution

Classified variants from ClinVar · 5 ACMG categories

ClassificationLoFMissense + InframeNon-codingSynonymousTotal
Pathogenic
0
0
50
0
50
Likely Pathogenic
0
0
5
0
5
VUS
0
54
11
0
65
Likely Benign
0
3
1
1
5
Benign
0
0
0
0
0
Total057671125

LoF = frameshift, stop gained/lost, canonical splice · Counts from ClinVar esearch · Updated hourly

View in ClinVar →

Protein Context — Lollipop Plot

GRK6 · protein map & ClinVar variants

Showing all ClinVar variants across the protein. Search a specific variant to highlight its position.

Clinical Trials

Active and recruiting trials from ClinicalTrials.gov

No active trials found for this gene.

Search ClinicalTrials.gov →
Clinical Literature
Open Research Assistant →