GOLGA2

Chr 9AR

golgin A2

Also known as: DEDHMB, GM130

This gene encodes a peripheral membrane protein that maintains Golgi apparatus structure, facilitates vesicle tethering and fusion, and plays essential roles in spindle pole assembly and centrosome organization during cell division. Mutations cause autosomal recessive developmental delays, intellectual disability, and skeletal abnormalities with onset in early childhood. The gene is highly constrained against loss-of-function variants (pLI 0.99, LOEUF 0.30), indicating that complete loss of protein function is likely incompatible with normal development.

OMIMResearchSummary from RefSeq, UniProt
ARLOEUF 0.291 OMIM phenotype
Clinical SummaryGOLGA2
Population Constraint (gnomAD)
Highly constrained gene — heterozygous loss-of-function variants are very rare in the population (pLI 0.99). One damaged copy is likely sufficient to cause disease.

Population Genetics & Constraint

gnomAD v4 — loss-of-function & missense intolerance

LoF intolerant — likely haploinsufficient
LoF Constraint
0.29LOEUF
pLI 0.988
Z-score 5.98
OE 0.18 (0.110.29)
Highly constrained

Highly LoF-intolerant (top ~10% of genes)

Missense Constraint
0.90Z-score
OE missense 0.89 (0.830.96)
520 obs / 581.2 exp
Tolerant

Mild missense constraint

Observed / Expected Ratios
LoF OE0.18 (0.110.29)
00.351.4
Missense OE0.89 (0.830.96)
00.61.4
Synonymous OE0.92
01.21.6
LoF obs/exp: 11 / 61.7Missense obs/exp: 520 / 581.2Syn Z: 0.91

ClinVar Variant Classifications

0 submitted variants in ClinVar

Protein Context — Lollipop Plot

GOLGA2 · protein map & ClinVar variants

Showing all ClinVar variants across the protein. Search a specific variant to highlight its position.

3D Protein StructureAlphaFold

Clinical Trials

Active and recruiting trials from ClinicalTrials.gov

No active trials found for this gene.

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Clinical Literature
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Recent Gene-Specific Literature
Gene in title · MEDLINE · newest first
Europe PMC