GLOD4

Chr 17

glyoxalase domain containing 4

Also known as: C17orf25, CGI-150, HC6, HC71

The protein enables cadherin binding activity and is located in extracellular exosomes and mitochondria. Mutations in this gene cause autosomal recessive developmental and epileptic encephalopathy with microcephaly. This gene is not highly constrained against loss-of-function variants, consistent with a recessive inheritance pattern.

OMIMResearchSummary from RefSeq
LOEUF 1.59
Clinical SummaryGLOD4
Population Constraint (gnomAD)
Low constraint (pLI 0.00) — loss-of-function variants are relatively tolerated in the population.

Population Genetics & Constraint

gnomAD v4 — loss-of-function & missense intolerance

Tolerant — LoF & missense variants common in population
LoF Constraint
1.59LOEUF
pLI 0.000
Z-score -0.30
OE 1.08 (0.741.59)
Tolerant

Highly tolerant — LoF variants common in population

Missense Constraint
0.01Z-score
OE missense 1.00 (0.881.13)
168 obs / 168.3 exp
Tolerant

Mild missense constraint

Observed / Expected Ratios
LoF OE1.08 (0.741.59)
00.351.4
Missense OE1.00 (0.881.13)
00.61.4
Synonymous OE1.11
01.21.6
LoF obs/exp: 18 / 16.7Missense obs/exp: 168 / 168.3Syn Z: -0.68

ClinVar Variant Classifications

0 submitted variants in ClinVar

Protein Context — Lollipop Plot

GLOD4 · protein map & ClinVar variants

Showing all ClinVar variants across the protein. Search a specific variant to highlight its position.

3D Protein StructureAlphaFold

Clinical Trials

Active and recruiting trials from ClinicalTrials.gov

No active trials found for this gene.

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