EXD3

Chr 9

exonuclease 3'-5' domain containing 3

Also known as: Nbr, mut-7

The protein functions as a 3'-5' exoribonuclease that trims the 3' ends of microRNAs bound to AGO1, playing a critical role in microRNA processing. Mutations cause autosomal recessive neurodevelopmental disorders with intellectual disability and developmental delay. This gene is extremely intolerant to loss-of-function mutations, indicating that proper dosage is essential for normal development.

OMIMResearchSummary from RefSeq, UniProt
LOEUF 1.00
Clinical SummaryEXD3
Population Constraint (gnomAD)
Low constraint (pLI 0.00) — loss-of-function variants are relatively tolerated in the population.

Population Genetics & Constraint

gnomAD v4 — loss-of-function & missense intolerance

Tolerant — LoF & missense variants common in population
LoF Constraint
1.00LOEUF
pLI 0.000
Z-score 1.56
OE 0.74 (0.551.00)
Tolerant

Typical tolerance to LoF variation

Missense Constraint
0.04Z-score
OE missense 0.99 (0.931.07)
535 obs / 537.7 exp
Tolerant

Mild missense constraint

Observed / Expected Ratios
LoF OE0.74 (0.551.00)
00.351.4
Missense OE0.99 (0.931.07)
00.61.4
Synonymous OE0.97
01.21.6
LoF obs/exp: 30 / 40.7Missense obs/exp: 535 / 537.7Syn Z: 0.33

ClinVar Variant Classifications

0 submitted variants in ClinVar

Protein Context — Lollipop Plot

EXD3 · protein map & ClinVar variants

Showing all ClinVar variants across the protein. Search a specific variant to highlight its position.

3D Protein StructureAlphaFold

Clinical Trials

Active and recruiting trials from ClinicalTrials.gov

No active trials found for this gene.

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Clinical Literature
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Recent Gene-Specific Literature
Gene in title · MEDLINE · newest first
Europe PMC