EIF3F

Chr 11AR

eukaryotic translation initiation factor 3 subunit F

Also known as: EIF3S5, MRT67, eIF3-p47

The EIF3F protein is a component of the eukaryotic translation initiation factor 3 complex, which is required for protein synthesis initiation by facilitating ribosome assembly and mRNA recruitment. Mutations cause autosomal recessive intellectual developmental disorder 67. This gene is highly constrained against loss-of-function mutations in the general population, indicating that such variants are likely to be pathogenic when present in the homozygous or compound heterozygous state.

OMIMResearchSummary from RefSeq, OMIM, UniProt
LOFmechanismARLOEUF 0.311 OMIM phenotype
Clinical SummaryEIF3F
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Gene-Disease Validity (ClinGen)
syndromic intellectual disability · ARDefinitive

Definitive — sufficient evidence for diagnostic panels

Population Constraint (gnomAD)
Highly constrained gene — heterozygous loss-of-function variants are very rare in the population (pLI 0.97). One damaged copy is likely sufficient to cause disease.
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Clinical Trials
1 active or recruiting trial — potential therapeutic options may be available

Population Genetics & Constraint

gnomAD v4 — loss-of-function & missense intolerance

LoF intolerant — likely haploinsufficient
LoF Constraint
0.31LOEUF
pLI 0.970
Z-score 3.39
OE 0.07 (0.020.31)
Highly constrained

Highly LoF-intolerant (top ~10% of genes)

Missense Constraint
-0.11Z-score
OE missense 1.02 (0.911.14)
212 obs / 207.6 exp
Tolerant

Tolerant to missense variation

Observed / Expected Ratios
LoF OE0.07 (0.020.31)
00.351.4
Missense OE1.02 (0.911.14)
00.61.4
Synonymous OE1.31
01.21.6
LoF obs/exp: 1 / 15.3Missense obs/exp: 212 / 207.6Syn Z: -2.24
Curated Mechanism (G2P)Gene2Phenotype (DDG2P) ↗
strongEIF3F-related developmental disorderLOFAR

Predictions shown for reference only — model trained on dominant genes, not applicable to AR conditions.

DN
0.4488th %ile
GOF
0.4875th %ile
LOF
0.65top 25%

The Badonyi & Marsh prediction model was trained exclusively on dominant disease genes. Predictions are not reliable for genes with autosomal recessive inheritance and are shown at reduced opacity for reference only.

Predictions from Badonyi M, Marsh JA. PLoS ONE. 2024;19(8):e0307312. Mechanism ranking also informed by gnomAD constraint, ClinVar, and ClinGen data.

ClinVar Variant Classifications

0 submitted variants in ClinVar

Protein Context — Lollipop Plot

EIF3F · protein map & ClinVar variants

Showing all ClinVar variants across the protein. Search a specific variant to highlight its position.

3D Protein StructureAlphaFold
Clinical Literature
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