COQ8A

Chr 1

coenzyme Q8A

Also known as: ADCK3, ARCA2, CABC1, COQ10D4, COQ8, SCAR9

The protein functions as an atypical kinase essential for coenzyme Q (ubiquinone) biosynthesis, which is required for mitochondrial electron transport and aerobic cellular respiration. Mutations cause primary coenzyme Q10 deficiency type 4, inherited in an autosomal recessive pattern. The pathogenic mechanism involves gain-of-function effects that disrupt normal coenzyme Q biosynthesis.

GeneReviewsResearchSummary from RefSeq, OMIM, UniProt, Mechanism
LOFmechanismLOEUF 1.01
Clinical SummaryCOQ8A
Population Constraint (gnomAD)
Low constraint (pLI 0.00) — loss-of-function variants are relatively tolerated in the population.
📋
ClinVar Variants
40 unique Pathogenic / Likely Pathogenic· 116 VUS of 300 total submissions
📖
GeneReview available — COQ8A
Authoritative clinical overview · Recommended first read
Open GeneReview ↗
Some data sources returned errors (1)

omim: Error: OMIM fetch failed: 429

Population Genetics & Constraint

gnomAD v4 — loss-of-function & missense intolerance

Tolerant — LoF & missense variants common in population
LoF Constraint
1.01LOEUF
pLI 0.000
Z-score 1.53
OE 0.69 (0.491.01)
Tolerant

Highly tolerant — LoF variants common in population

Missense Constraint
-0.87Z-score
OE missense 1.12 (1.041.21)
448 obs / 399.0 exp
Tolerant

Tolerant to missense variation

Observed / Expected Ratios
LoF OE0.69 (0.491.01)
00.351.4
Missense OE1.12 (1.041.21)
00.61.4
Synonymous OE1.27
01.21.6
LoF obs/exp: 20 / 28.9Missense obs/exp: 448 / 399.0Syn Z: -2.76
Curated Mechanism (G2P)Gene2Phenotype (DDG2P) ↗
definitiveCOQ8A-related coenzyme Q10 deficiencyLOFAR

Predictions shown for reference only — model trained on dominant genes, not applicable to AR conditions.

DN
0.6746th %ile
GOF
0.6541th %ile
LOF
0.3358th %ile

The Badonyi & Marsh prediction model was trained exclusively on dominant disease genes. Predictions are not reliable for genes with autosomal recessive inheritance and are shown at reduced opacity for reference only.

Predictions from Badonyi M, Marsh JA. PLoS ONE. 2024;19(8):e0307312.

ClinVar Variant Classifications

300 submitted variants in ClinVar

Classification Summary

Pathogenic19
Likely Pathogenic21
VUS116
Likely Benign123
Benign3
Conflicting3
19
Pathogenic
21
Likely Pathogenic
116
VUS
123
Likely Benign
3
Benign
3
Conflicting

Curated Variants Distribution

Classified variants from ClinVar · 5 ACMG categories

ClassificationLoFMissense + InframeNon-codingSynonymousTotal
Pathogenic
14
2
3
0
19
Likely Pathogenic
13
8
0
0
21
VUS
2
98
14
2
116
Likely Benign
0
1
49
73
123
Benign
0
0
3
0
3
Conflicting
3
Total291096975285

LoF = frameshift, stop gained/lost, canonical splice · Counts from ClinVar esearch · Updated hourly

View in ClinVar →

Protein Context — Lollipop Plot

COQ8A · protein map & ClinVar variants

Showing all ClinVar variants across the protein. Search a specific variant to highlight its position.

3D Protein StructureAlphaFold

Clinical Trials

Active and recruiting trials from ClinicalTrials.gov

No active trials found for this gene.

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Clinical Literature
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Full-Text Mentions
NLP-detected gene mentions in article bodies · via PubTator3
PubTator3
Top 5 full-text resultsSearch PubTator3 ↗