COLEC10

Chr 8AR

collectin subfamily member 10

Also known as: 3MC3, CL-10, CL-34, CLL1

This gene encodes a member of the C-lectin family, proteins that possess collagen-like sequences and carbohydrate recognition domains. The other members of this family are secreted proteins and bind to carbohydrate antigens on microorganisms facilitating their recognition and removal. This gene product is a cytosolic protein, a characteristic that suggests that it may have different biological functions than other C-lectins. [provided by RefSeq, Jul 2008]

Primary Disease Associations & Inheritance

3MC syndrome 3MIM #248340
AR
6
Active trials
9
Pubs (1 yr)
0
P/LP submissions
P/LP missense
0.96
LOEUF
LOF
Mechanism· G2P
Clinical SummaryCOLEC10
Population Constraint (gnomAD)
Low constraint (pLI 0.00) — loss-of-function variants are relatively tolerated in the population.
💊
Clinical Trials
6 active or recruiting trials — potential therapeutic options may be available

Population Genetics & Constraint

gnomAD v4 — loss-of-function & missense intolerance

Tolerant — LoF & missense variants common in population
LoF Constraint
0.96LOEUF
pLI 0.003
Z-score 1.66
OE 0.49 (0.270.96)
Tolerant

Typical tolerance to LoF variation

Missense Constraint
0.03Z-score
OE missense 0.99 (0.871.13)
158 obs / 159.0 exp
Tolerant

Mild missense constraint

Observed / Expected Ratios
LoF OE0.49 (0.270.96)
00.351.4
Missense OE0.99 (0.871.13)
00.61.4
Synonymous OE0.88
01.21.6
LoF obs/exp: 6 / 12.3Missense obs/exp: 158 / 159.0Syn Z: 0.71
Curated Mechanism (G2P)Gene2Phenotype (DDG2P) ↗
strongCOLEC10-related 3MC syndromeLOFAR

Predictions shown for reference only — model trained on dominant genes, not applicable to AR conditions.

DN
0.7033th %ile
GOF
0.6442th %ile
LOF
0.3067th %ile

The Badonyi & Marsh prediction model was trained exclusively on dominant disease genes. Predictions are not reliable for genes with autosomal recessive inheritance and are shown at reduced opacity for reference only.

Predictions from Badonyi M, Marsh JA. PLoS ONE. 2024;19(8):e0307312.

ClinVar Variant Classifications

0 submitted variants in ClinVar

Protein Context — Lollipop Plot

COLEC10 · protein map & ClinVar variants

Showing all ClinVar variants across the protein. Search a specific variant to highlight its position.

Clinical Trials

Active and recruiting trials from ClinicalTrials.gov

Acute Myeloid Leukemia

A Clinical Study to Evaluate the Safety and Efficacy of CLL1 and CD38 Dual-Target CAR-T Cell Injection in the Treatment of Relapsed or Refractory Acute Myeloid Leukemia

RECRUITING
NCT06880354Phase PHASE1Institute of Hematology & Blood Diseases Hospital, ChinaStarted 2025-05-22
CLL1 and CD38 Dual-Target CAR-T Cell Injection
Acute Myeloid LeukemiaChimeric Antigen Receptor T-cellUniversal CLL1 CAR-T

Clinical Study on the Safety, Efficacy and Pharmacokinetics of Universal CLL1 Chimeric Antigen Receptor T-Cell in Relapsed/Refractory Acute Myeloid Leukemia

NOT YET RECRUITING
NCT07432100Phase PHASE1Mingfeng ZhaoStarted 2026-02
universal CLL1 CAR-T cells
AML (Acute Myeloid Leukemia)CAR-T

Clinical Study of CLL-1 CAR-T in the Treatment of Children With R/R AML

NOT YET RECRUITING
NCT07338357Phase EARLY_PHASE1First Affiliated Hospital of Guangxi Medical UniversityStarted 2026-01-05
CAR-T
LeukemiaAcute Myeloid Leukemia

BG1805 Injection in the Treatment of Relapsed or Refractory Acute Myeloid Leukemia in Children

NOT YET RECRUITING
NCT06347458Phase PHASE1Guangzhou Bio-gene Technology Co., LtdStarted 2024-04
BG1805
Leukemia, Myeloid, Acute

Phase I Clinical Study: BG1805 Injection in the Treatment of Relapsed or Refractory Acute Myeloid Leukemia

RECRUITING
NCT06118788Phase PHASE1, PHASE2Guangzhou Bio-gene Technology Co., LtdStarted 2024-03-07
BG1805
Acute Myeloid Leukemia

Chimeric Antigen Receptor T-cells for The Treatment of AML Expressing CLL-1 Antigen

ACTIVE NOT RECRUITING
NCT04219163Phase PHASE1Baylor College of MedicineStarted 2020-07-09
CLL-1.CAR T cells
Clinical Literature
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