CDADC1

Chr 13

cytidine and dCMP deaminase domain containing 1

Also known as: NYD-SP15, bA103J18.1

The protein catalyzes the deamination of cytidine and deoxycytidine into uridine and deoxyuridine, respectively, and may play an important role in testicular development and spermatogenesis. Mutations cause autosomal recessive primary immunodeficiency with defective lymphocyte apoptosis and autoimmunity. This gene is extremely intolerant to loss-of-function variants, suggesting complete loss of protein function is likely incompatible with normal development.

OMIMResearchSummary from RefSeq, UniProt
LOEUF 1.10
Clinical SummaryCDADC1
Population Constraint (gnomAD)
Low constraint (pLI 0.00) — loss-of-function variants are relatively tolerated in the population.

Population Genetics & Constraint

gnomAD v4 — loss-of-function & missense intolerance

Tolerant — LoF & missense variants common in population
LoF Constraint
1.10LOEUF
pLI 0.000
Z-score 1.18
OE 0.74 (0.511.10)
Tolerant

Highly tolerant — LoF variants common in population

Missense Constraint
1.47Z-score
OE missense 0.75 (0.670.84)
202 obs / 270.1 exp
Tolerant

Mild missense constraint

Observed / Expected Ratios
LoF OE0.74 (0.511.10)
00.351.4
Missense OE0.75 (0.670.84)
00.61.4
Synonymous OE0.93
01.21.6
LoF obs/exp: 18 / 24.3Missense obs/exp: 202 / 270.1Syn Z: 0.57

ClinVar Variant Classifications

0 submitted variants in ClinVar

Protein Context — Lollipop Plot

CDADC1 · protein map & ClinVar variants

Showing all ClinVar variants across the protein. Search a specific variant to highlight its position.

3D Protein StructureAlphaFold

Clinical Trials

Active and recruiting trials from ClinicalTrials.gov

No active trials found for this gene.

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