CALML3

Chr 10

calmodulin like 3

May function as a specific light chain of unconventional myosin-10 (MYO10), also enhances MYO10 translation, possibly by acting as a chaperone for the emerging MYO10 heavy chain protein. May compete with calmodulin by binding, with different affinities, to cellular substrates

0
Active trials
8
Pubs (1 yr)
0
P/LP submissions
P/LP missense
1.90
LOEUF
DN
Mechanism· predicted
Clinical SummaryCALML3
Population Constraint (gnomAD)
Low constraint (pLI 0.00) — loss-of-function variants are relatively tolerated in the population.
Some data sources returned errors (1)

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Population Genetics & Constraint

gnomAD v4 — loss-of-function & missense intolerance

Tolerant — LoF & missense variants common in population
LoF Constraint
1.90LOEUF
pLI 0.001
Z-score -0.43
OE 1.26 (0.571.90)
Tolerant

Highly tolerant — LoF variants common in population

Missense Constraint
0.94Z-score
OE missense 0.76 (0.640.90)
90 obs / 118.7 exp
Tolerant

Mild missense constraint

Observed / Expected Ratios
LoF OE1.26 (0.571.90)
00.351.4
Missense OE0.76 (0.640.90)
00.61.4
Synonymous OE0.82
01.21.6
LoF obs/exp: 4 / 3.2Missense obs/exp: 90 / 118.7Syn Z: 1.05
DN
0.86top 5%
GOF
0.5758th %ile
LOF
0.2189th %ile

The highest-scoring mechanism for this gene is dominant-negative.

DNprediction above median

Note: In-silico variant effect predictors (SIFT, PolyPhen, REVEL, CADD) may underestimate pathogenicity of missense variants in genes with GOF or DN mechanisms. Consider functional evidence and clinical context.

Predictions from Badonyi M, Marsh JA. PLoS ONE. 2024;19(8):e0307312.

ClinVar Variant Classifications

0 submitted variants in ClinVar

Protein Context — Lollipop Plot

CALML3 · protein map & ClinVar variants

Showing all ClinVar variants across the protein. Search a specific variant to highlight its position.

Clinical Trials

Active and recruiting trials from ClinicalTrials.gov

No active trials found for this gene.

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Clinical Literature
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