CACNG7

Chr 19

calcium voltage-gated channel auxiliary subunit gamma 7

The protein regulates AMPA-selective glutamate receptor trafficking and gating properties, specifically for GRIA1 and GRIA2 subunits, and also modulates L-type calcium channels containing CACNA1C. Mutations cause autosomal dominant cone-rod dystrophy, an inherited retinal degeneration affecting central vision that typically presents in childhood or adolescence. The gene is highly constrained against loss-of-function variation, indicating that such mutations are likely to be pathogenic.

OMIMResearchSummary from RefSeq, UniProt
LOEUF 0.41
Clinical SummaryCACNG7
Population Constraint (gnomAD)
Moderately constrained gene (pLI 0.89) — some intolerance to loss-of-function variants.

Population Genetics & Constraint

gnomAD v4 — loss-of-function & missense intolerance

Moderate LoF intolerance
LoF Constraint
0.41LOEUF
pLI 0.887
Z-score 2.87
OE 0.09 (0.030.41)
Moderately constrained

More LoF-intolerant than ~75% of genes

Missense Constraint
1.65Z-score
OE missense 0.66 (0.570.77)
124 obs / 187.4 exp
Tolerant

Mild missense constraint

Observed / Expected Ratios
LoF OE0.09 (0.030.41)
00.351.4
Missense OE0.66 (0.570.77)
00.61.4
Synonymous OE0.94
01.21.6
LoF obs/exp: 1 / 11.5Missense obs/exp: 124 / 187.4Syn Z: 0.46

ClinVar Variant Classifications

0 submitted variants in ClinVar

Protein Context — Lollipop Plot

CACNG7 · protein map & ClinVar variants

Showing all ClinVar variants across the protein. Search a specific variant to highlight its position.

3D Protein StructureAlphaFold

Clinical Trials

Active and recruiting trials from ClinicalTrials.gov

No active trials found for this gene.

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