C3ORF52

Chr 3AR

chromosome 3 open reading frame 52

Also known as: HYPT15, TTMP

This protein facilitates LIPH-mediated synthesis of 2-acyl lysophosphatidic acid (LPA), a bioactive lipid mediator necessary for hair formation and growth. Mutations cause hypotrichosis 15, an autosomal recessive condition characterized by sparse hair development. The gene shows low constraint to loss-of-function variation (pLI 0.00009, LOEUF 1.8), consistent with recessive inheritance where heterozygous carriers are typically unaffected.

OMIMResearchSummary from RefSeq, OMIM, UniProt
ARLOEUF 1.791 OMIM phenotype
Clinical SummaryC3ORF52
Population Constraint (gnomAD)
Low constraint (pLI 0.00) — loss-of-function variants are relatively tolerated in the population.

Population Genetics & Constraint

gnomAD v4 — loss-of-function & missense intolerance

Tolerant — LoF & missense variants common in population
LoF Constraint
1.79LOEUF
pLI 0.000
Z-score -0.06
OE 1.03 (0.551.79)
Tolerant

Highly tolerant — LoF variants common in population

Missense Constraint
0.86Z-score
OE missense 0.78 (0.660.93)
97 obs / 123.8 exp
Tolerant

Mild missense constraint

Observed / Expected Ratios
LoF OE1.03 (0.551.79)
00.351.4
Missense OE0.78 (0.660.93)
00.61.4
Synonymous OE0.75
01.21.6
LoF obs/exp: 6 / 5.9Missense obs/exp: 97 / 123.8Syn Z: 1.41

ClinVar Variant Classifications

0 submitted variants in ClinVar

Protein Context — Lollipop Plot

C3ORF52 · protein map & ClinVar variants

Showing all ClinVar variants across the protein. Search a specific variant to highlight its position.

3D Protein StructureAlphaFold

Clinical Trials

Active and recruiting trials from ClinicalTrials.gov

No active trials found for this gene.

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Clinical Literature
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Recent Gene-Specific Literature
Gene in title · MEDLINE · newest first
Europe PMC