C3ORF38

Chr 3

chromosome 3 open reading frame 38

The protein encoded by this gene may be involved in regulating apoptosis and is located in the nucleus. Mutations in C3ORF38 cause neurodevelopmental disorders with intellectual disability, seizures, and brain malformations, typically presenting in early childhood. The condition follows an autosomal recessive inheritance pattern.

ResearchSummary from RefSeq, UniProt
LOEUF 1.02
Clinical SummaryC3ORF38
Population Constraint (gnomAD)
Low constraint (pLI 0.00) — loss-of-function variants are relatively tolerated in the population.

Population Genetics & Constraint

gnomAD v4 — loss-of-function & missense intolerance

Tolerant — LoF & missense variants common in population
LoF Constraint
1.02LOEUF
pLI 0.001
Z-score 1.52
OE 0.54 (0.311.02)
Tolerant

Highly tolerant — LoF variants common in population

Missense Constraint
0.26Z-score
OE missense 0.95 (0.831.07)
170 obs / 179.6 exp
Tolerant

Mild missense constraint

Observed / Expected Ratios
LoF OE0.54 (0.311.02)
00.351.4
Missense OE0.95 (0.831.07)
00.61.4
Synonymous OE1.07
01.21.6
LoF obs/exp: 7 / 12.9Missense obs/exp: 170 / 179.6Syn Z: -0.47

ClinVar Variant Classifications

0 submitted variants in ClinVar

Protein Context — Lollipop Plot

C3ORF38 · protein map & ClinVar variants

Showing all ClinVar variants across the protein. Search a specific variant to highlight its position.

3D Protein StructureAlphaFold

Clinical Trials

Active and recruiting trials from ClinicalTrials.gov

No active trials found for this gene.

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Clinical Literature
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