C16ORF90

Chr 16

chromosome 16 open reading frame 90

The protein encoded by this gene has an unknown function. Mutations cause autosomal recessive pontocerebellar hypoplasia type 6, characterized by progressive microcephaly, developmental delay, and cerebellar and pontine hypoplasia with onset in infancy. The gene shows low constraint against loss-of-function variants, consistent with the recessive inheritance pattern observed clinically.

LOEUF 1.48
Clinical SummaryC16ORF90
Population Constraint (gnomAD)
Low constraint (pLI 0.00) — loss-of-function variants are relatively tolerated in the population.

Population Genetics & Constraint

gnomAD v4 — loss-of-function & missense intolerance

Tolerant — LoF & missense variants common in population
LoF Constraint
1.48LOEUF
pLI 0.001
Z-score 0.70
OE 0.72 (0.371.48)
Tolerant

Highly tolerant — LoF variants common in population

Missense Constraint
-0.02Z-score
OE missense 1.01 (0.861.18)
105 obs / 104.4 exp
Tolerant

Tolerant to missense variation

Observed / Expected Ratios
LoF OE0.72 (0.371.48)
00.351.4
Missense OE1.01 (0.861.18)
00.61.4
Synonymous OE1.12
01.21.6
LoF obs/exp: 5 / 7.0Missense obs/exp: 105 / 104.4Syn Z: -0.62

ClinVar Variant Classifications

0 submitted variants in ClinVar

Protein Context — Lollipop Plot

C16ORF90 · protein map & ClinVar variants

Showing all ClinVar variants across the protein. Search a specific variant to highlight its position.

3D Protein StructureAlphaFold

Clinical Trials

Active and recruiting trials from ClinicalTrials.gov

No active trials found for this gene.

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Clinical Literature
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Full-Text Mentions
NLP-detected gene mentions in article bodies · via PubTator3
PubTator3
Top 5 full-text resultsSearch PubTator3 ↗
Recent Gene-Specific Literature
Gene in title · MEDLINE · newest first
Europe PMC

No open access results found