ANKRD39

Chr 2

ankyrin repeat domain 39

ANKRD39 encodes an ankyrin repeat domain-containing protein that functions in protein-protein interactions and cellular signaling pathways. Mutations cause autosomal recessive developmental and epileptic encephalopathy with severe intellectual disability and seizures beginning in infancy. The gene shows low constraint against loss-of-function variants, consistent with its recessive inheritance pattern.

0
Active trials
0
Pubs (1 yr)
31
P/LP submissions
0%
P/LP missense
1.21
LOEUF
Multiple*
Mechanism· predicted
Clinical SummaryANKRD39
Population Constraint (gnomAD)
Low constraint (pLI 0.00) — loss-of-function variants are relatively tolerated in the population.
📋
ClinVar Variants
31 unique Pathogenic / Likely Pathogenic· 74 VUS of 112 total submissions

Population Genetics & Constraint

gnomAD v4 — loss-of-function & missense intolerance

Tolerant — LoF & missense variants common in population
LoF Constraint
1.21LOEUF
pLI 0.001
Z-score 1.13
OE 0.61 (0.331.21)
Tolerant

Highly tolerant — LoF variants common in population

Missense Constraint
0.18Z-score
OE missense 0.95 (0.821.11)
112 obs / 117.5 exp
Tolerant

Mild missense constraint

Observed / Expected Ratios
LoF OE0.61 (0.331.21)
00.351.4
Missense OE0.95 (0.821.11)
00.61.4
Synonymous OE0.86
01.21.6
LoF obs/exp: 6 / 9.8Missense obs/exp: 112 / 117.5Syn Z: 0.81
DN
0.6648th %ile
GOF
0.6443th %ile
LOF
0.3068th %ile

This gene has evidence for multiple mechanisms of pathogenicity (dominant-negative and gain-of-function). Both the Badonyi & Marsh prediction and the broader genomic evidence point to dominant-negative as the predominant mechanism. Different variants in this gene may act through different mechanisms — interpret in context of the specific variant.

DNprediction above median
GOFprediction above median

Note: In-silico variant effect predictors (SIFT, PolyPhen, REVEL, CADD) may underestimate pathogenicity of missense variants in genes with GOF or DN mechanisms. Consider functional evidence and clinical context.

Predictions from Badonyi M, Marsh JA. PLoS ONE. 2024;19(8):e0307312.

ClinVar Variant Classifications

112 submitted variants in ClinVar

Classification Summary

Pathogenic21
Likely Pathogenic10
VUS74
Likely Benign4
21
Pathogenic
10
Likely Pathogenic
74
VUS
4
Likely Benign

Curated Variants Distribution

Classified variants from ClinVar · 5 ACMG categories

ClassificationLoFMissense + InframeNon-codingSynonymousTotal
Pathogenic
0
0
21
0
21
Likely Pathogenic
0
0
10
0
10
VUS
0
44
30
0
74
Likely Benign
0
1
2
1
4
Benign
0
0
0
0
0
Total045631109

LoF = frameshift, stop gained/lost, canonical splice · Counts from ClinVar esearch · Updated hourly

View in ClinVar →

Protein Context — Lollipop Plot

ANKRD39 · protein map & ClinVar variants

Showing all ClinVar variants across the protein. Search a specific variant to highlight its position.

Clinical Trials

Active and recruiting trials from ClinicalTrials.gov

No active trials found for this gene.

Search ClinicalTrials.gov →
Clinical Literature
Open Research Assistant →