ANKRD36C

Chr 2

ankyrin repeat domain 36C

The ANKRD36C protein is predicted to function as an ion channel inhibitor. Mutations in this gene have not been definitively associated with any recognized human diseases or clinical phenotypes in the current medical literature. The inheritance pattern and clinical significance of ANKRD36C variants remain to be established.

ResearchSummary from RefSeq
Multiplemechanism

Population Genetics & Constraint

Constraint data not available from gnomAD.

DN
0.77top 25%
GOF
0.77top 25%
LOF
0.2969th %ile

This gene has evidence for multiple mechanisms of pathogenicity (dominant-negative and gain-of-function). Both the Badonyi & Marsh prediction and the broader genomic evidence point to dominant-negative as the predominant mechanism. Different variants in this gene may act through different mechanisms — interpret in context of the specific variant.

DNprediction above median
GOFprediction above median

Note: In-silico variant effect predictors (SIFT, PolyPhen, REVEL, CADD) may underestimate pathogenicity of missense variants in genes with GOF or DN mechanisms. Consider functional evidence and clinical context.

Predictions from Badonyi M, Marsh JA. PLoS ONE. 2024;19(8):e0307312.

ClinVar Variant Classifications

0 submitted variants in ClinVar

Protein Context — Lollipop Plot

ANKRD36C · protein map & ClinVar variants

Showing all ClinVar variants across the protein. Search a specific variant to highlight its position.

3D Protein StructureAlphaFold

Clinical Trials

Active and recruiting trials from ClinicalTrials.gov

No active trials found for this gene.

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