AJM1

Chr 9

apical junction component 1 homolog

Also known as: C9orf172, ajm-1

The protein is predicted to control adherens junction integrity and organize cell-cell junctions at apical plasma membranes and ciliary structures. This gene is highly constrained against loss-of-function variants (pLI 0.99, LOEUF 0.29), suggesting that mutations likely cause severe developmental disorders, though specific associated diseases have not yet been established. Inheritance pattern has not been determined.

OMIMResearchSummary from RefSeq, UniProt
LOEUF 0.28
Clinical SummaryAJM1
Population Constraint (gnomAD)
Highly constrained gene — heterozygous loss-of-function variants are very rare in the population (pLI 0.99). One damaged copy is likely sufficient to cause disease.

Population Genetics & Constraint

gnomAD v4 — loss-of-function & missense intolerance

LoF intolerant — likely haploinsufficient
LoF Constraint
0.28LOEUF
pLI 0.988
Z-score 3.96
OE 0.09 (0.040.28)
Highly constrained

Highly LoF-intolerant (top ~10% of genes)

Missense Constraint
2.80Z-score
OE missense 0.67 (0.610.73)
378 obs / 565.6 exp
Mild constraint

Moderately missense-constrained (top ~2.5%)

Observed / Expected Ratios
LoF OE0.09 (0.040.28)
00.351.4
Missense OE0.67 (0.610.73)
00.61.4
Synonymous OE1.01
01.21.6
LoF obs/exp: 2 / 22.1Missense obs/exp: 378 / 565.6Syn Z: -0.12

ClinVar Variant Classifications

0 submitted variants in ClinVar

Protein Context — Lollipop Plot

AJM1 · protein map & ClinVar variants

Showing all ClinVar variants across the protein. Search a specific variant to highlight its position.

3D Protein StructureAlphaFold

Clinical Trials

Active and recruiting trials from ClinicalTrials.gov

No active trials found for this gene.

Search ClinicalTrials.gov →
Clinical Literature
Open Research Assistant →
Recent Gene-Specific Literature
Gene in title · MEDLINE · newest first
Europe PMC

No open access results found