AGXT

Chr 2AR

alanine--glyoxylate aminotransferase

The protein is a peroxisomal aminotransferase that catalyzes the transamination of glyoxylate to glycine, contributing to glyoxylate detoxification in the liver. Mutations cause primary hyperoxaluria type 1, an autosomal recessive disorder that leads to excessive oxalate production and can result in kidney stones, nephrocalcinosis, and progressive kidney disease. This gene is not constrained against loss-of-function variants, and some disease-causing mutations are known to alter the protein's subcellular targeting.

OMIMResearchSummary from RefSeq, OMIM, UniProt
LOFmechanismARLOEUF 1.031 OMIM phenotype
Clinical SummaryAGXT
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Gene-Disease Validity (ClinGen)
alanine glyoxylate aminotransferase deficiency · ARDefinitive

Definitive — sufficient evidence for diagnostic panels

Population Constraint (gnomAD)
Low constraint (pLI 0.00) — loss-of-function variants are relatively tolerated in the population.
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Clinical Trials
6 active or recruiting trials — potential therapeutic options may be available
Some data sources returned errors (1)

ncbi: Error: NCBI fetch failed: 429 https://eutils.ncbi.nlm.nih.gov/entrez/eutils/esearch.fcgi

Population Genetics & Constraint

gnomAD v4 — loss-of-function & missense intolerance

Tolerant — LoF & missense variants common in population
LoF Constraint
1.03LOEUF
pLI 0.000
Z-score 1.46
OE 0.66 (0.431.03)
Tolerant

Highly tolerant — LoF variants common in population

Missense Constraint
-0.27Z-score
OE missense 1.05 (0.951.16)
263 obs / 251.1 exp
Tolerant

Tolerant to missense variation

Observed / Expected Ratios
LoF OE0.66 (0.431.03)
00.351.4
Missense OE1.05 (0.951.16)
00.61.4
Synonymous OE1.08
01.21.6
LoF obs/exp: 14 / 21.3Missense obs/exp: 263 / 251.1Syn Z: -0.64
Curated Mechanism (G2P)Gene2Phenotype (DDG2P) ↗
definitiveAGXT-related hyperoxaluria, primaryLOFAR

Predictions shown for reference only — model trained on dominant genes, not applicable to AR conditions.

DN
0.74top 25%
GOF
0.5759th %ile
LOF
0.3164th %ile

The Badonyi & Marsh prediction model was trained exclusively on dominant disease genes. Predictions are not reliable for genes with autosomal recessive inheritance and are shown at reduced opacity for reference only.

Predictions from Badonyi M, Marsh JA. PLoS ONE. 2024;19(8):e0307312.

ClinVar Variant Classifications

0 submitted variants in ClinVar

Protein Context — Lollipop Plot

AGXT · protein map & ClinVar variants

Showing all ClinVar variants across the protein. Search a specific variant to highlight its position.

3D Protein StructureAlphaFold
Clinical Literature
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